自噬介导胰腺肿瘤诱发细胞在缺氧微环境中存活》的更正。

IF 5.6 2区医学 Q1 ONCOLOGY

The Journal of Pathology Pub Date : 2024-03-21 DOI:10.1002/path.6281

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引用次数: 0

摘要

Vanessa Rausch, Li Liu, Anja Apel, Theresa Rettig, Jury Gladkich, Sabrina Labsch, Georgios Kallifatidis, Adam Kaczorowski, Ariane Groth, Wolfgang Gross, Martha M Gebhard, Peter Schemmer, Jens Werner, Alexei V Salnikov, Hanswalter Zentgraf, Markus W Büchler and Ingrid Herr.J Pathol 2012; 227: 325-335. https://doi.org/10.1002/path.3994 本文于 2012 年 1 月 19 日首次发表于 Wiley Online Library (wileyonlinelibrary.com)，通讯作者告知编辑本文图 2E 中的一处错误。作者对其错误及造成的不便深表歉意。在检查其档案并与编辑和出版商讨论其发现后，作者希望再做两处修改：图 4B 第三行中也使用了这些对照β-肌动蛋白条带，这在科学上是有效的。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

Correction to ‘Autophagy mediates survival of pancreatic tumour-initiating cells in a hypoxic microenvironment’

查看原文本刊更多论文

Correction to ‘Autophagy mediates survival of pancreatic tumour-initiating cells in a hypoxic microenvironment’

The corresponding author informed the editors of an error in Figure 2E of this article, first published on 19 January 2012 in Wiley Online Library (wileyonlinelibrary.com).

The authors apologize for their error and the inconvenience caused.

After examining their archive and discussing their findings with the editors and publisher, the author wished to make two additional changes:

These control β-actin bands were used also in the third row of Figure 4B, and this was scientifically valid.

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来源期刊

The Journal of Pathology 医学-病理学

CiteScore

14.10

自引率

1.40%

发文量

144

审稿时长

3-8 weeks

期刊介绍： The Journal of Pathology aims to serve as a translational bridge between basic biomedical science and clinical medicine with particular emphasis on, but not restricted to, tissue based studies. The main interests of the Journal lie in publishing studies that further our understanding the pathophysiological and pathogenetic mechanisms of human disease. The Journal of Pathology welcomes investigative studies on human tissues, in vitro and in vivo experimental studies, and investigations based on animal models with a clear relevance to human disease, including transgenic systems. As well as original research papers, the Journal seeks to provide rapid publication in a variety of other formats, including editorials, review articles, commentaries and perspectives and other features, both contributed and solicited.