CDK4/6抑制剂Palbociclib与ATRA协同诱导急性髓细胞白血病患者的分化。

IF 5.3 2区 医学 Q1 ONCOLOGY
Linhui Hu, Qian Li, Jiyu Wang, Huiping Wang, Xiyang Ren, Keke Huang, Yangyang Wang, Xue Liang, Lianfang Pu, Shudao Xiong, Zhimin Zhai
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引用次数: 0

摘要

基于 ATRA 的分化疗法几乎治愈了急性早幼粒细胞白血病(APL)。然而,令人失望的是,ATRA 对其他急性髓性白血病(AML)亚型无效。开发促进白血病分化的新型有效抗 AML 疗法非常必要。CDK4/6-cyclin D通路是决定细胞命运的G1/S期转变的关键启动器。在此,我们研究了CDK4/6抑制剂palbociclib是否能与ATRA协同促进体外和体内的白血病分化。我们的研究结果表明,CDK4/6-环素D通路基因在急性髓细胞白血病中异常表达,而且我们观察到,palbociclib能使急性髓细胞白血病细胞对ATRA诱导的形态、生化和功能变化敏感,这些变化表明了髓细胞的分化。palbociclib和ATRA联合使用可减轻AML细胞在体内的扩增。这些增强的分化效应可能与 RARα、E2F1 和 STAT1 等转录因子的调控有关。总之,我们的研究结果表明,CDK4/6抑制可使AML细胞对ATRA敏感,并可指导AML患者新型治疗策略的开发。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
The CDK4/6 Inhibitor Palbociclib Synergizes with ATRA to Induce Differentiation in AML.

Differentiation therapy based on ATRA almost cured acute promyelocytic leukemia (APL). However, it is disappointing that ATRA is not effective against other acute myeloid leukemia (AML) subtypes. Developing new and effective anti-AML therapies that promote leukemia differentiation is necessary. The CDK4/6-cyclin D pathway is a key initiator of the G1-S phase transition, which determines cell fate. Herein, we investigated whether the CDK4/6 inhibitor palbociclib would synergize with ATRA to promote leukemia differentiation in vitro and in vivo. Our findings revealed that CDK4/6-cyclin D pathway genes were aberrantly expressed in AML, and we observed that palbociclib sensitized AML cells to ATRA-induced morphologic, biochemical, and functional changes indicative of myeloid differentiation. The combination of palbociclib and ATRA attenuated AML cell expansion in vivo. These enhanced differentiation effects may be associated with the regulation of transcription factors, including RARα, E2F1, and STAT1. Overall, our findings demonstrate that CDK4/6 inhibition sensitizes AML cells to ATRA and could guide the development of novel therapeutic strategies for patients with AML.

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来源期刊
CiteScore
11.20
自引率
1.80%
发文量
331
审稿时长
3 months
期刊介绍: Molecular Cancer Therapeutics will focus on basic research that has implications for cancer therapeutics in the following areas: Experimental Cancer Therapeutics, Identification of Molecular Targets, Targets for Chemoprevention, New Models, Cancer Chemistry and Drug Discovery, Molecular and Cellular Pharmacology, Molecular Classification of Tumors, and Bioinformatics and Computational Molecular Biology. The journal provides a publication forum for these emerging disciplines that is focused specifically on cancer research. Papers are stringently reviewed and only those that report results of novel, timely, and significant research and meet high standards of scientific merit will be accepted for publication.
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