硒酶Ⅰ型碘甲状腺原氨酸脱碘酶:卵巢癌中一种新的肿瘤抑制因子。

IF 6.6 2区 医学 Q1 Biochemistry, Genetics and Molecular Biology
Molecular Oncology Pub Date : 2024-09-01 Epub Date: 2024-03-01 DOI:10.1002/1878-0261.13612
Adi Alfandari, Dotan Moskovich, Avivit Weisz, Aviva Katzav, Debora Kidron, Mario Beiner, Dana Josephy, Aula Asali, Yael Hants, Yael Yagur, Omer Weitzner, Martin Ellis, Gilad Itchaki, Osnat Ashur-Fabian
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引用次数: 0

摘要

硒酶Ⅰ型碘甲状腺原氨酸脱碘酶(DIO1)催化甲状腺激素中碘原子的脱除。尽管有报道称 DIO1 的作用在多种恶性肿瘤中受到干扰,但在致死率最高的妇科癌症--高级别浆液性卵巢癌(HGSOC)中还没有开展任何研究。我们研究了 DIO1 在 HGSOC 患者(癌症基因组图谱(TCGA)数据和肿瘤组织)、人细胞系(ES-2 和 Kuramochi)、正常中国仓鼠卵巢细胞(CHO-K1)和正常人输卵管细胞(FT282 和 FT109)中的表达。为了研究其功能作用,对 DIO1 进行了过表达、抑制(丙基硫脲嘧啶(PTU))或敲除(KD),并进行了细胞计数、增殖、凋亡、细胞活力和蛋白质组学分析。与正常细胞和组织相比,在 HGSOC 中观察到较低的 DIO1 水平。TCGA分析证实,DIO1 mRNA的低表达与患者的生存率和耐药性相关。沉默或抑制该酶会导致卵巢癌增殖增强,而异位表达 DIO1 则会产生相反的效果。DIO1-KD细胞中的蛋白质组学分析表明,促进肿瘤代谢和进展的蛋白质发生了全面变化。总之,DIO1的表达与卵巢癌的进展成反比,这表明这种酶具有抑制肿瘤的作用,并有可能用作这种疾病的生物标记物。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
The selenoenzyme type I iodothyronine deiodinase: a new tumor suppressor in ovarian cancer.

The selenoenzyme type I iodothyronine deiodinase (DIO1) catalyzes removal of iodine atoms from thyroid hormones. Although DIO1 action is reported to be disturbed in several malignancies, no work has been conducted in high-grade serous ovarian carcinoma (HGSOC), the most lethal gynecologic cancer. We studied DIO1 expression in HGSOC patients [The Cancer Genome Atlas (TCGA) data and tumor tissues], human cell lines (ES-2 and Kuramochi), normal Chinese hamster ovarian cells (CHO-K1), and normal human fallopian tube cells (FT282 and FT109). To study its functional role, DIO1 was overexpressed, inhibited [by propylthiouracil (PTU)], or knocked down (KD), and cell count, proliferation, apoptosis, cell viability, and proteomics analysis were performed. Lower DIO1 levels were observed in HGSOC compared to normal cells and tissues. TCGA analyses confirmed that low DIO1 mRNA expression correlated with worse survival and therapy resistance in patients. Silencing or inhibiting the enzyme led to enhanced ovarian cancer proliferation, while an opposite effect was shown following DIO1 ectopic expression. Proteomics analysis in DIO1-KD cells revealed global changes in proteins that facilitate tumor metabolism and progression. In conclusion, DIO1 expression and ovarian cancer progression are inversely correlated, highlighting a tumor suppressive role for this enzyme and its potential use as a biomarker in this disease.

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来源期刊
Molecular Oncology
Molecular Oncology Biochemistry, Genetics and Molecular Biology-Molecular Medicine
CiteScore
11.80
自引率
1.50%
发文量
203
审稿时长
10 weeks
期刊介绍: Molecular Oncology highlights new discoveries, approaches, and technical developments, in basic, clinical and discovery-driven translational cancer research. It publishes research articles, reviews (by invitation only), and timely science policy articles. The journal is now fully Open Access with all articles published over the past 10 years freely available.
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