自闭症的发病机制和分子差异

Jingwen Xie
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引用次数: 0

摘要

随着人们对自闭症认识的加深,自闭症谱系障碍(ASD)的范围也在不断扩大。全球自闭症的发病率已超过 1%。语言和社会沟通问题,以及反复出现的刻板兴趣和行为,是自闭症的主要表现。为了探究自闭症的发病机理,科学家们已经发现了很多证据,表明自闭症是一种由多种因素导致的复杂疾病,但仍有很多问题没有得到解答。有关自闭症病因和症状的论文不胜枚举,但往往只有少数观点受到关注。本综述旨在总结自闭症的致病因素,包括基因和环境因素,并列出自闭症在基因、蛋白质和细胞方面与正常人有何不同。本综述将通过一些实例来介绍这些因素。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
The Pathogenesis and Differences in molecule of Autism
With the deepening of the understanding of autism, the scope of autism spectrum disorder (ASD) continues to expand. The prevalence rate of ASD has arrived over 1% around the world. Language and social communication issues, as well as recurrent stereotyped interests and behaviors, are the main signs of autism. To find out the pathogenesis of autism, scientists have found a lot, which show that autism is a complex disorder resulting from many factors, but many questions remain unanswered. Numerous theses have been written about the causes and symptoms of autism, but frequently only a few of these perspectives have received attention. This review is to sum up the causative factors of autism, including genes and environmental, and lists how autism different from normal people in terms of genes, proteins, and cells. This review will introduce these factors with some examples.
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