严重创伤导致的心力衰竭

Maximilian Dietrich, Frank Weilbacher, Stephan Katzenschlager, Markus A. Weigand, Erik Popp
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引用次数: 0

摘要

尽管我们在加强创伤护理方面做出了巨大努力,但创伤性心脏骤停(TCA)的死亡率仍然非常高。因此,我院采取了特别措施来优化重大创伤患者的治疗。这些措施包括院前医疗干预车(MIC)和创伤复苏室针对 TCA 或休克患者的 "红色代码 "协议。这些措施能够及早治疗 TCA 的可逆性病因,并使大量患者获得充分的 ROSC。然而,这些患者中仍有相当一部分在不久后因循环衰竭而死亡。根据我们对接受瓣膜胸廓切开术或超声心动图评估的患者的观察,并结合当前的科学发现,我们得出结论:心脏功能障碍本身可能就是原因。因此,我们建议将严重创伤相关性心力衰竭(STAC)作为一个新的实体进行讨论,以促进科学研究和特定治疗策略的开发,从而改善严重创伤的预后。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Severe trauma associated cardiac failure
Although significant efforts have been made to enhance trauma care, the mortality rate for traumatic cardiac arrest (TCA) remains exceedingly high. Therefore, our institution has implemented special measures to optimize the treatment of major trauma patients. These measures include a prehospital Medical Intervention Car (MIC) and a ‘code red’ protocol in the trauma resuscitation room for patients with TCA or shock. These measures enable the early treatment of reversible causes of TCA and have resulted in a significant number of patients achieving adequate ROSC. However, a significant proportion of these patients still die due to circulatory failure shortly after. Our observations from patients who underwent clamshell thoracotomy or received echocardiographic evaluation in conjunction with current scientific findings led us to conclude that dysfunction of the heart itself may be the cause. Therefore, we propose discussing severe trauma-associated cardiac failure (STAC) as a new entity to facilitate scientific research and the development of specific treatment strategies, with the aim of improving the outcome of severe trauma.
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