病例报告:1例急性慢性肝衰竭合并罕见肝性脑病及不明原因发热患者经人工肝支持系统后抗感染联合心理治疗完全缓解

Yan Bo, Rengaowa Sha, Yuanjie Jiang, Haodong Yu
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摘要

本报告的主题是不可思议的心理治疗对不明原因发热(FUO)的启发。我们从心理免疫、肝脏免疫、炎症、病因、诊断方法和治疗策略等方面分析了这一令人难以置信的现象。男,44岁,因腹胀皮肤、巩膜发黄染色40多天,羽毛伴其4天入院。肝炎病毒测试显示HBV感染。HBV DNA检测呈阴性。呼吸道九倍、肺结核、贫血、肠俱乐部比检测均为阴性。由于病人身体虚弱,我们无法配合更多的检查。我们用血样监测和分析了25天。总之,这是一个慢性急性肝功能衰竭、狂躁、发热的病例,从治疗方案、一般治疗、人工肝治疗、手术治疗、抗感染治疗、心理治疗等方面来看,可能是FUO引起的。经过25天的治疗,患者达到了常见的治疗目标。本报告旨在为慢性急性肝功能衰竭、躁狂、FUO患者提供心理治疗方案和临床特点,并为长期血液标本检查提供启发性思考策略。心理发热的潜在机制是免疫功能紊乱,免疫球蛋白A、E浓度下降可能是直接因素。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Case Report: Complete Remission of a Patient with Acute-on-chronic Liver Failure with Rare Hepatic Encephalopathy and Fever of Unknown Origin Treated with Anti-infective Therapy Combined with Psychotherapy After Artificial Liver Support System
The theme of this report is incredible psychological therapy's inspiration for fever of unknown origin (FUO). We analyzed this incredible phenomenon through psychological immunity, liver immunity, inflammation, etiology, diagnostic methods, and treatment strategies. A man, 44 years old, was dyed for more than 40 days due to abdominal distension with skin and scleral yellow, and feathers accompanied him for four days to enter the hospital. Hepatitis virus test shows HBV infection. HBV DNA detection shows negative. The detection of the respiratory tract nine-fold, tuberculosis, anemia, and gut-to-club ratio all show negative. Due to the weakness of the patient's body, we cannot cooperate with more examinations. We use blood specimens to monitor and analyze for 25 days. In short, this is a case of chronic-acute liver failure, manic, and fever, likely to be an FUO cause in terms of the treatment plan, general treatment, artificial liver treatment, surgical treatment, anti-infection therapy, and psychotherapy. The patient reached the common treatment goal after 25 days of treatment. This report aims to provide psychotherapy programs and clinical characteristics for patients with chronic-acute liver failure, manic, and FUO and provide inspirational thinking strategies for long-term blood specimen examination. The potential mechanism of psychological fever is immune disorders, and the decline in the concentration of immunoglobulin A and E may be a direct factor.
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