Analytic studies on fetal heart rate changes to prevent cerebral palsy with novel hypoxia index in fetal monitoring

Fetal heart rate (FHR) rises when the fetus moves, where FHR acceleration is reacted fetal brain to fetal movements (burst), it losts in early fetal hypoxia, while FHR variability is the reaction to minor fetal motions, which losts in severe hypoxia followed by cerebral palsy. FHR falls in hypoxia when fetal PaO2 is 50 or less mmHg, while fetal PaO2 is lower than 50 mmHg, where fetal vagal nerve center is excited by hypoxia, forming FHR bradycardia and deceleration. The late deceleration was ominous in the past, while 3 connective typical late decelerations’ outcome was normal, while repeated late deceleraions for 50 minutes developed heavy asphyxia and severe brain damage, thus, novel hypoxia index is the sum of deceleration durations (min) divided by the lowest FHR, and multiplied by 100, where no cerebral palsy developed when hypoxia index was 24 or less, while the index was 25 or more in cases of cerebral palsy. Visual FHR patttern classification will be changed to objective hypoxia index and FHR score which predicts Apgar score and UApH. FHR frequency spectrum diagnoses pathologic sinusoidal FHR.caused by severe fetal anemia imminent to fetal death. *Correspondence to: Kazuo Maeda, MD, PhD, 3-125 Nadamachi, Yonago, Tottoriken, 683-0835, Japan, Tel: +81859226856; E-mail: maedak@mocha.ocn.ne.jp