开颅减压术的并发症

Andrew C. Margules, Jack Jallo
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引用次数: 1

摘要

颅内高压的处理通常是神经外科医生治疗严重创伤性脑损伤(TBI)患者的最大挑战。颅内压(ICP)的急剧升高会使大脑调节脑血流量(CBF)的能力不堪重负。由此引起的脑缺血可导致功能减退或死亡。常规治疗方案,如高渗性脱水、过度通气和巴比妥酸昏迷,是第一道防线。然而,据报道,10-15%由严重脑外伤引起的颅内高压患者对最大限度的医疗管理没有反应在这些难治性病例中,减压颅骨切除术可能提供挽救生命的ICP降低。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Complications of Decompressive Craniectomy
Introduction Management of intracranial hypertension is often the greatest challenge for a neurosurgeon treating a patient who has suffered a severe traumatic brain injury (TBI). Too sharp a rise in intracranial pressure (ICP) can overwhelm the brain’s ability to regulate cerebral blood flow (CBF). The resulting cerebral ischemia can contribute to diminished function or to death. Conventional treatment options, such as hyperosmolar dehydration, hyperventilation and barbiturate coma, form the first line of defense. However, it has been reported that 10-15% of patients with intracranial hypertension resulting from severe TBI do not respond to maximal medical management.1 In these refractory cases, decompressive craniectomy may provide a lifesaving reduction in ICP.
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