[闪现诱发视觉电位在颅面骨折视神经损伤早期诊断中的应用]。

E Altenmüller, C P Cornelius, H Uhl
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引用次数: 0

摘要

约10%的颅面骨折患者因视神经损伤而出现视力障碍或丧失。视神经功能的评估是决定视神经减压的重要依据。但检查视力和瞳孔反射可能是困难的,特别是在意识下降和原发性瞳孔功能障碍的不合作患者。在这些情况下,视神经功能可以通过使用led护目镜刺激器引发的闪烁诱发视觉电位来监测。我们记录了10例伴有长时间意识改变的颅面骨折和脑震荡的头部损伤患者的vep。在入院时的急性期进行录音。恢复意识后检查视敏度和视野,临床表现与初始vep的相关性。经临床检查,4例初始vep正常的患者双眼视力正常。1例患者最初表现为单侧vep幅度降低50%以上,临床表现为同心视野缺损。3例单侧电位丧失患者为单侧无症状。在没有vep的一名患者中保留了对光的感知。一名双侧电位丧失患者在恢复意识后失明。一般来说,入学时瞳孔光反射测试与vep结果相符。然而,在两个病例中,瞳孔反应性丧失,但vep仍然存在。这些患者视力正常,但瞳孔反射传出通路受损。在另外两名患者中,由于眼睑极度水肿,无法进行瞳孔反应性检查。(摘要删节250字)
本文章由计算机程序翻译,如有差异,请以英文原文为准。
[Flash-evoked visual potentials in the early diagnosis of optic nerve injury due to craniofacial fractures].

Impairment or loss of vision due to optic nerve injury occurs in about 10% of patients with cranio-facial fractures. The assessment of optic nerve function is important for decisions regarding optic nerve decompression. But examination of vision and pupillary reflexes may be difficult, especially in uncooperative patients with reduced consciousness and primary disturbances of pupillary functions. In these cases, optic nerve function can be monitored by means of flash-evoked visual potentials elicited by use of a LED-goggle stimulator. VEPs were recorded in ten patients with head injuries comprising cranio-facial fractures and cerebral concussion with prolonged alteration of consciousness. Recordings were obtained in the acute phase upon admission. Visual acuity and visual fields were examined after regaining consciousness and the clinical findings correlated to the initial VEPs. Upon clinical examinations, four patients with initially normal VEPs had normal vision on both eyes. One patient initially revealed unilateral reduction of the VEP-amplitude of more than 50% and clinically showed a concentric visual field defect. Three patients with unilateral loss of potentials were amaurotic on this side. Perception of light was preserved in one patient in whom VEPs were absent. One patient with bilateral loss of potentials was blind when consciousness was regained. In general, pupillary light reflexes tested at admission corresponded to the VEP-findings. In two cases, however, pupillary reactivity was lost, but VEPs were still present. These patients had normal vision, but exhibited a lesion of the efferent pathways of pupillary reflexes. In two other patients, examination of pupillary reactivity could not be performed due to extreme edema of the eyelids.(ABSTRACT TRUNCATED AT 250 WORDS)

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