[电解质紊乱纠正后高血压患者血流动力学变化的各种神经体液方面]。

Sovetskaia meditsina Pub Date : 1991-01-01
G M Isazade, R F Gadzhiev, A B Bakhshaliev
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引用次数: 0

摘要

本文研究了83例高血压I期和II期患者在考虑“盐敏感性”的情况下,纠正钠平衡时交感肾上腺和+肾素-血管紧张素-醛固酮系统(SAS和RAAS)活性、血流动力学和电解质代谢的变化。在适度限制氯化钠时,患者的反应并不均匀。“盐敏感”受试者反应积极,中枢和外周血流动力学改善,电解质平衡,SAS和RAAS活性增强。在盐无反应和矛盾敏感性中,SAS和RAAS表现为激活,在矛盾敏感性中最明显,中枢和外周血流动力学更差,电解质失衡。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
[Various neurohumoral aspects of hemodynamic changes in patients with hypertension after correction of electrolyte disorders].

Changes in activity of sympathetic-adrenal and + renin-angiotensin-aldosterone systems (SAS and RAAS), hemodynamics and electrolyte metabolism when correcting sodium balance in consideration of their "salt sensitivity" were studied in 83 hypertension stage I and II patients. In moderate restriction of sodium chloride response of the patients was not uniform. "Salt--sensitive" subjects responded positively with improvement of central and peripheral hemodynamics, electrolyte balance in enhanced activity of SAS and RAAS. In salt nonresponders and in paradoxical sensitivity SAS and RAAS show activation, most distinct in paradoxical sensitivity, and worse central and peripheral hemodynamics, electrolyte imbalance.

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