使用的文学

B. C. Albensi, Mattson, Anderson, J. Holtz, G. Baskin, P. P. Wang, S. L. Menzaghi, Z. C. Baquet, J. Górski, K. R. Jones, C. Barnes, G. Rao, B. L. Mcnaughton
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引用次数: 29

摘要

(1999):“老年大鼠脑脊液α -分泌酶裂解淀粉样蛋白水平降低:与空间记忆缺陷的相关性”,神经科学93:1409 - 1420。(2004):“缺乏顺行性皮层脑源性神经营养因子的早期纹状体树突缺损与老年神经元丧失”,《神经科学杂志》24,4250—4258。海马体:细胞兴奋性变化的可能机制[J] .中华神经科学杂志,2009,549 - 558。人类齿状回突触连接区在衰老和阿尔茨海默病中的适应性反应[j],《脑科学进展》517,69 - 75。突触大小在衰老和老年痴呆中的代偿性扩大>,in: Boll Soc Ital Biol Sper 69, 57 - 63。和/或trkC神经营养因子受体引起老年海马和杏仁核的结构改变[J] .欧洲神经科学杂志18,2319‐2325。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Verwendete Literatur
(1999): »Reduced cerebrospinal fluid levels of alpha‐secretase‐cleaved amyloid precursor protein in aged rats: correlation with spatial memory deficits«, in: Neurosci 93: 1409‐1420. (2004): »Early striatal dendrite deficits followed by neuron loss with advanced age in the absence of anterograde cortical brain‐derived neurotrophic factor«, in: J Neurosci 24, 4250‐4258. hippocampus: a possible mechanism for cellular excitability changes«, in: J Comp Neurol 259, 549‐558. adaptive response of the synaptic junctional zones in the human dentate gyrus during aging and Alzheimer's disease«, in: Brain Res 517, 69‐75. »Compensatory enlargement of synaptic size in aging and senile dementia«, in: Boll Soc Ital Biol Sper 69, 57‐63. and/or trkC neurotrophin receptors causes structural alterations in the aged hippocampus and amygdala«, in: Eur J Neurosci 18, 2319‐2325.
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