{"title":"放射科医师对评估腭咽功能不全的贡献","authors":"Donald Harrigan","doi":"10.1055/s-0028-1095238","DOIUrl":null,"url":null,"abstract":"Normal speech and voice production depend in large measure upon separation of the oral and nasal cavities by closure of the velopharyngeal port. This is accomplished by elevation of the velum or soft palate and the sphincteric action of the pharyngeal walls. T h e elevation and posterior movement of the soft palate, which occurs as a result of the combined contributions of the levator palatini and the palatopharyngeus muscles, gives the most important contribution to closure and is easily and clearly demonstrated by radiological techniques. Medial movement of the lateral pharyngeal walls against the edges of the velum also assists in closure of the lateral aspects of the velopharyngeal portal. The forward bulging of the posterior pharyngeal wall in normal speech is slight and its contribution to speech is probably insignificant. However, pharyngeal wall motion may become significant in the individual with palate dysfunction. Lateral pharyngeal wall motion can be studied by radiological techniques (Skolnick, 1969) as well as by ultrasound (Kelsey et al., 1972). There must be synchronous motion of both components of the closure mechanism to prevent the nasal emission of sound. Failure of the velopharyngeal portal to close during the utterence of non-nasal sounds is referred to as velopharyngeal insufficiency. Velopharyngeal insufficiency can be caused by a number of factors, including congenital structural abnormalities, such as a cleft or shortened palate, congenital or acquired neuromuscular dysfunction, or traumatically induced clefts. Although data are available on the frequency of maxillofacial clefts, there are no good data available on the frequency of velopharyngeal insufficiency from all causes. Evaluation of the velopharyngeal port for degree and location of incompetence requires visualization and/or physiological monitoring during a wide range of speech tasks. The re are a number of techniques available, which can be divided into invasive or noninvasive categories (Table 1). All invasive techniques, with the possi-","PeriodicalId":364385,"journal":{"name":"Seminars in Speech, Language and Hearing","volume":"23 1","pages":"0"},"PeriodicalIF":0.0000,"publicationDate":"1982-08-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"1","resultStr":"{\"title\":\"The Contribution of the Radiologist to Evaluation of Velopharyngeal Insufficiency\",\"authors\":\"Donald Harrigan\",\"doi\":\"10.1055/s-0028-1095238\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"Normal speech and voice production depend in large measure upon separation of the oral and nasal cavities by closure of the velopharyngeal port. This is accomplished by elevation of the velum or soft palate and the sphincteric action of the pharyngeal walls. T h e elevation and posterior movement of the soft palate, which occurs as a result of the combined contributions of the levator palatini and the palatopharyngeus muscles, gives the most important contribution to closure and is easily and clearly demonstrated by radiological techniques. Medial movement of the lateral pharyngeal walls against the edges of the velum also assists in closure of the lateral aspects of the velopharyngeal portal. The forward bulging of the posterior pharyngeal wall in normal speech is slight and its contribution to speech is probably insignificant. However, pharyngeal wall motion may become significant in the individual with palate dysfunction. Lateral pharyngeal wall motion can be studied by radiological techniques (Skolnick, 1969) as well as by ultrasound (Kelsey et al., 1972). There must be synchronous motion of both components of the closure mechanism to prevent the nasal emission of sound. Failure of the velopharyngeal portal to close during the utterence of non-nasal sounds is referred to as velopharyngeal insufficiency. Velopharyngeal insufficiency can be caused by a number of factors, including congenital structural abnormalities, such as a cleft or shortened palate, congenital or acquired neuromuscular dysfunction, or traumatically induced clefts. Although data are available on the frequency of maxillofacial clefts, there are no good data available on the frequency of velopharyngeal insufficiency from all causes. Evaluation of the velopharyngeal port for degree and location of incompetence requires visualization and/or physiological monitoring during a wide range of speech tasks. The re are a number of techniques available, which can be divided into invasive or noninvasive categories (Table 1). 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引用次数: 1
摘要
正常的言语和声音的产生在很大程度上依赖于通过关闭腭咽口来分离口腔和鼻腔。这是通过腭膜或软腭的抬高和咽壁的括约肌作用来完成的。由于提腭肌和腭咽肌的共同作用,软腭的抬高和后移对闭合起着最重要的作用,放射技术可以很容易地、清楚地证明这一点。咽侧壁在膜边缘的内侧运动也有助于咽侧壁门的闭合。正常说话时咽后壁的前凸是轻微的,它对说话的贡献可能是微不足道的。然而,在腭功能障碍的个体中,咽壁运动可能变得明显。可以通过放射技术(Skolnick, 1969)和超声波(Kelsey et al., 1972)来研究咽壁外侧运动。关闭机构的两个组成部分必须同步运动,以防止鼻腔发出声音。在发出非鼻音时,腭咽门不能关闭被称为腭咽功能不全。腭咽功能不全可由多种因素引起,包括先天性结构异常,如腭裂或腭裂缩短,先天性或获得性神经肌肉功能障碍,或创伤性腭裂。虽然有关于颌面裂的频率的数据,但没有关于所有原因引起的腭咽功能不全的频率的良好数据。评估腭咽口功能不全的程度和位置需要在广泛的言语任务中进行可视化和/或生理监测。有许多可用的技术,可分为侵入性和非侵入性两类(表1)。所有侵入性技术,都有可能
The Contribution of the Radiologist to Evaluation of Velopharyngeal Insufficiency
Normal speech and voice production depend in large measure upon separation of the oral and nasal cavities by closure of the velopharyngeal port. This is accomplished by elevation of the velum or soft palate and the sphincteric action of the pharyngeal walls. T h e elevation and posterior movement of the soft palate, which occurs as a result of the combined contributions of the levator palatini and the palatopharyngeus muscles, gives the most important contribution to closure and is easily and clearly demonstrated by radiological techniques. Medial movement of the lateral pharyngeal walls against the edges of the velum also assists in closure of the lateral aspects of the velopharyngeal portal. The forward bulging of the posterior pharyngeal wall in normal speech is slight and its contribution to speech is probably insignificant. However, pharyngeal wall motion may become significant in the individual with palate dysfunction. Lateral pharyngeal wall motion can be studied by radiological techniques (Skolnick, 1969) as well as by ultrasound (Kelsey et al., 1972). There must be synchronous motion of both components of the closure mechanism to prevent the nasal emission of sound. Failure of the velopharyngeal portal to close during the utterence of non-nasal sounds is referred to as velopharyngeal insufficiency. Velopharyngeal insufficiency can be caused by a number of factors, including congenital structural abnormalities, such as a cleft or shortened palate, congenital or acquired neuromuscular dysfunction, or traumatically induced clefts. Although data are available on the frequency of maxillofacial clefts, there are no good data available on the frequency of velopharyngeal insufficiency from all causes. Evaluation of the velopharyngeal port for degree and location of incompetence requires visualization and/or physiological monitoring during a wide range of speech tasks. The re are a number of techniques available, which can be divided into invasive or noninvasive categories (Table 1). All invasive techniques, with the possi-