儿童跗骨舟骨骨坏死

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引用次数: 3

摘要

儿童跗骨舟骨骨坏死(无血管性、无菌性或缺血性骨坏死)可能自发发生(原发性、特发性、非创伤性或非创伤性),也可能继发于创伤(创伤后)和骨软骨病。在原发性和继发性骨坏死的两组中,临床表现和影像学异常都是自限性的,通常自发消退,与负重和固定治疗方式无关。基于在无症状和有症状的儿童中检测到的类似的硬化和变平的x线表现,Köhler的疾病被定义为非外伤性舟骨坏死或骨软骨硬化过程。儿童创伤后跗舟骨坏死可继发于微创伤或过度使用性损伤、应力性骨折、急性骨折、剥离性骨软骨炎和严重的足部损伤。这篇社论旨在介绍儿童跗骨舟骨坏死的主要和次要原因,并描述临床和影像学评估的困难,以便确定准确的诊断。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Tarsal Navicular Osteonecrosis in Children
Osteonecrosis (avascular, aseptic or ischemic bone necrosis) of the tarsal navicular in children may develop either spontaneously (primary, idiopathic, atraumatic or non-traumatic) or secondary to trauma (post-traumatic) and osteochondrosis. In both groups, of primary and secondary osteonecrosis, the clinical findings as well as the radiographic abnormalities are self-limited and usually resolve spontaneously irrespective of weight-bearing and immobilization treatment modalities. Köhler’s disease has been defined either as atraumatic navicular osteonecrosis or as an osteochondrosis process, based on the similar radiographic appearance of increased sclerosis and flattening detected in both asymptomatic and symptomatic children. Post-traumatic tarsal navicular osteonecrosis in children may follow microtrauma or overuse injuries, stress fractures, acute fractures, osteochondritis dissecans and severe foot injuries. This editorial aims to present the primary and secondary causes of osteonecrosis of the tarsal navicular in children and to describe the difficulties of the clinical and radiological evaluation in order to define an accurate diagnosis.
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