{"title":"冠状动脉事件的复发:内皮功能障碍作为中介","authors":"Fiama Caimi, Bruno Strada","doi":"10.29277/cardio.35.1.16","DOIUrl":null,"url":null,"abstract":"There is evidence that after a coronary event, an inflammatory response is generated, increasing the endothelium dysfunction, which may motivate a new short-term coronary event. Recurrence of ischemic phenomena is described in the vessel involved in the initial event and other vascular territories. Potentially therapeutic physiopathological modifications do not have a clinical impact applicable to usual cardiological practice.","PeriodicalId":191575,"journal":{"name":"Revista Uruguaya de Cardiología","volume":"19 1","pages":"0"},"PeriodicalIF":0.0000,"publicationDate":"2020-03-17","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Reincidencia de eventos coronarios: disfunción endotelial como mediadora\",\"authors\":\"Fiama Caimi, Bruno Strada\",\"doi\":\"10.29277/cardio.35.1.16\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"There is evidence that after a coronary event, an inflammatory response is generated, increasing the endothelium dysfunction, which may motivate a new short-term coronary event. Recurrence of ischemic phenomena is described in the vessel involved in the initial event and other vascular territories. Potentially therapeutic physiopathological modifications do not have a clinical impact applicable to usual cardiological practice.\",\"PeriodicalId\":191575,\"journal\":{\"name\":\"Revista Uruguaya de Cardiología\",\"volume\":\"19 1\",\"pages\":\"0\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2020-03-17\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Revista Uruguaya de Cardiología\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.29277/cardio.35.1.16\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Revista Uruguaya de Cardiología","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.29277/cardio.35.1.16","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Reincidencia de eventos coronarios: disfunción endotelial como mediadora
There is evidence that after a coronary event, an inflammatory response is generated, increasing the endothelium dysfunction, which may motivate a new short-term coronary event. Recurrence of ischemic phenomena is described in the vessel involved in the initial event and other vascular territories. Potentially therapeutic physiopathological modifications do not have a clinical impact applicable to usual cardiological practice.