S. Meena, M. Mittal, R. Yadav, Anju Ranga, M. Sinha
{"title":"双侧膝状体出血性梗死——胰腺炎的罕见并发症","authors":"S. Meena, M. Mittal, R. Yadav, Anju Ranga, M. Sinha","doi":"10.25259/crcr_7_2022","DOIUrl":null,"url":null,"abstract":"We report a case of 10-year-male child diagnosed as acute pancreatitis with deranged liver and renal functions, who presented with sudden onset bilateral painless vision loss 2 days after initial diagnosis. Neuroophthalmological examination including deep tendon reflex, plantar reflex, visual acuity, pupillary reflexes, and fundus was performed. Magnetic resonance imaging brain demonstrated features of hemorrhagic infarction involving bilateral lateral geniculate bodies (LGBs) and posterior end of optic tracts. Two major causes of bilateral LGBs lesions include vascular ischemia and osmotic demyelination. Possible mechanism of involvement of bilateral LGBs in present case is coagulopathy in the settings of acute pancreatitis attributed to pro-thrombotic state leading to microvascular occlusions.","PeriodicalId":419021,"journal":{"name":"Case Reports in Clinical Radiology","volume":"7 1","pages":"0"},"PeriodicalIF":0.0000,"publicationDate":"2023-01-17","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Hemorrhagic infarction of bilateral lateral geniculate bodies – unusual complication of pancreatitis\",\"authors\":\"S. Meena, M. Mittal, R. Yadav, Anju Ranga, M. Sinha\",\"doi\":\"10.25259/crcr_7_2022\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"We report a case of 10-year-male child diagnosed as acute pancreatitis with deranged liver and renal functions, who presented with sudden onset bilateral painless vision loss 2 days after initial diagnosis. Neuroophthalmological examination including deep tendon reflex, plantar reflex, visual acuity, pupillary reflexes, and fundus was performed. Magnetic resonance imaging brain demonstrated features of hemorrhagic infarction involving bilateral lateral geniculate bodies (LGBs) and posterior end of optic tracts. Two major causes of bilateral LGBs lesions include vascular ischemia and osmotic demyelination. Possible mechanism of involvement of bilateral LGBs in present case is coagulopathy in the settings of acute pancreatitis attributed to pro-thrombotic state leading to microvascular occlusions.\",\"PeriodicalId\":419021,\"journal\":{\"name\":\"Case Reports in Clinical Radiology\",\"volume\":\"7 1\",\"pages\":\"0\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2023-01-17\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Case Reports in Clinical Radiology\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.25259/crcr_7_2022\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Case Reports in Clinical Radiology","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.25259/crcr_7_2022","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Hemorrhagic infarction of bilateral lateral geniculate bodies – unusual complication of pancreatitis
We report a case of 10-year-male child diagnosed as acute pancreatitis with deranged liver and renal functions, who presented with sudden onset bilateral painless vision loss 2 days after initial diagnosis. Neuroophthalmological examination including deep tendon reflex, plantar reflex, visual acuity, pupillary reflexes, and fundus was performed. Magnetic resonance imaging brain demonstrated features of hemorrhagic infarction involving bilateral lateral geniculate bodies (LGBs) and posterior end of optic tracts. Two major causes of bilateral LGBs lesions include vascular ischemia and osmotic demyelination. Possible mechanism of involvement of bilateral LGBs in present case is coagulopathy in the settings of acute pancreatitis attributed to pro-thrombotic state leading to microvascular occlusions.