突发性感音神经性听力损失1例报告

Fikrilah Abdul Azis, Afiya Shafa Kamilah, Arifiana Larasati Restyani, Athaya Miraghassani, Henny Widyastuti
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摘要

背景:突发性感音神经性听力损失(SSNHL)通常被称为突发性耳聋,是一种无法解释的快速听力损失,要么是一次性的,要么是在几天内,患者的听力突然下降。它通常与感染、头部创伤、自身免疫性疾病、医源性、严重感染、血液循环问题、神经系统疾病(如多发性硬化症)、内耳疾病(如姆氏病)、甚至代谢紊乱(如糖尿病)有关。我们介绍了印度尼西亚一家二级医院的突发性感音神经性听力损失病例,以增加对该主题的更多了解。病例介绍男性,76岁,主诉为右侧突发性听力丧失。他没有其他症状,耳朵也没有外伤史。患者有高血压,定期服用氨氯地平和可乐定。患者耳镜、鼻镜检查结果正常。听力学显示左耳阈值为37 dB,右耳阈值为117 dB,空气和骨均进行纯音刺激,双耳鼓室图均为a型。诊断为右耳突发性感音神经性听力损失,给予甲泼尼龙、阿昔洛韦、甲钴胺、维生素B6、雷尼替丁、奥美拉唑治疗。结论本病的病理生理机制为迷路病毒感染、迷路血管受损、耳蜗内膜破裂和免疫介导的内耳疾病。在我们的病例中,我们怀疑缺血性血管疾病和病毒感染都是导致ISHHL的原因。如果没有明确的或可治疗的病因,治疗方案应该由最可能的因素决定,在我们的病例中是全身性类固醇和阿昔洛韦。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Sudden Sensorineural Hearing Loss (SSNHL): Case Report
Background Sudden sensorineural hearing loss (SSNHL) commonly known as sudden deafness, is an unexplained, rapid loss of hearing either all at once or over a few days in which patients experience a sudden drop in hearing. It is commonly linked to infections, head trauma, autoimmune diseases, iatrogenic, severe infections, blood circulation problems, neurological disorders, such as multiple sclerosis, disorders of the inner ear, such as Ménière’s disease, and even metabolic disorders such as diabetes mellitus. We presented a case of sudden sensorineural hearing loss in a secondary hospital in Indonesia to add more knowledge on this topic. Case presentation A 76-year-old male, had chief complain of a sudden right sided hearing loss. There were no other symptoms and he had no history of trauma to the ear. The patient had hypertension and consumed amlodipine and clonidine regularly. Patient had normal otoscopic and rhinoscopy examination results. Audiometry shown a hearing threshold of 37 dB in the left ear and 117 dB in the right ear, both in air and bone conducted pure-tone stimuli and tympanogram showed type As in both ears. A diagnosis of right ear sudden sensorineural hearing loss was made, and medical treatment consisted of methylprednisolone, acyclovir, mecobalamin, vitamin B6, ranitidine and omeprazole were given to the patient. Conclusion Pathophysiology of this disease are labyrinthine viral infection, labyrinthine vascular compromise, intracochlear membrane ruptures, and immune-mediated inner ear disease. In our case, we suspect that both ischemic vascular disease and viral infection as the cause of ISHHL. If there is no definitive or treatable etiology found, treatment regimen should be dictated by the most likely factors involved, which were systemic steroids and acyclovir in our case.
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