{"title":"脑膜炎球菌败血症中的隔室综合征","authors":"A. Rahoma, A. Malek","doi":"10.5580/11b9","DOIUrl":null,"url":null,"abstract":"The clinical syndrome results from the activation and continued stimulation of the immune system by proinflammatory cytokines. This process is caused directly by bacterial components, such as endotoxins released from the bacterial cell wall, and indirectly by the activation of inflammatory cells.. Combined, the processes produce multiorgan failure that usually causes cardiorespiratory depression and, possibly, renal, neurological, and gastrointestinal failure.","PeriodicalId":284795,"journal":{"name":"The Internet Journal of Plastic Surgery","volume":"6 1","pages":"0"},"PeriodicalIF":0.0000,"publicationDate":"2006-12-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"1","resultStr":"{\"title\":\"Compartment Syndrome In Meningiococcal Septicemia\",\"authors\":\"A. Rahoma, A. Malek\",\"doi\":\"10.5580/11b9\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"The clinical syndrome results from the activation and continued stimulation of the immune system by proinflammatory cytokines. This process is caused directly by bacterial components, such as endotoxins released from the bacterial cell wall, and indirectly by the activation of inflammatory cells.. Combined, the processes produce multiorgan failure that usually causes cardiorespiratory depression and, possibly, renal, neurological, and gastrointestinal failure.\",\"PeriodicalId\":284795,\"journal\":{\"name\":\"The Internet Journal of Plastic Surgery\",\"volume\":\"6 1\",\"pages\":\"0\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2006-12-31\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"1\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"The Internet Journal of Plastic Surgery\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.5580/11b9\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"The Internet Journal of Plastic Surgery","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.5580/11b9","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
The clinical syndrome results from the activation and continued stimulation of the immune system by proinflammatory cytokines. This process is caused directly by bacterial components, such as endotoxins released from the bacterial cell wall, and indirectly by the activation of inflammatory cells.. Combined, the processes produce multiorgan failure that usually causes cardiorespiratory depression and, possibly, renal, neurological, and gastrointestinal failure.