{"title":"肿瘤诱导的骨溶解和高钙血症的机制。","authors":"M Dowsett","doi":"","DOIUrl":null,"url":null,"abstract":"<p><p>This is a brief resume of the current knowledge regarding the mechanisms whereby tumours may cause the breakdown or increase the turnover of bone matrix resulting in hypercalcaemia. The precise mechanisms and agents still remain to be ascertained. This would appear to be an investigative area worthy of significant research effort in view of the potential benefit to patient care which may then ensue.</p>","PeriodicalId":75950,"journal":{"name":"Investigative & cell pathology","volume":"1 2","pages":"159-70"},"PeriodicalIF":0.0000,"publicationDate":"1978-04-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"The mechanisms of human tumour-induced osteolysis and hypercalcaemia.\",\"authors\":\"M Dowsett\",\"doi\":\"\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p><p>This is a brief resume of the current knowledge regarding the mechanisms whereby tumours may cause the breakdown or increase the turnover of bone matrix resulting in hypercalcaemia. The precise mechanisms and agents still remain to be ascertained. This would appear to be an investigative area worthy of significant research effort in view of the potential benefit to patient care which may then ensue.</p>\",\"PeriodicalId\":75950,\"journal\":{\"name\":\"Investigative & cell pathology\",\"volume\":\"1 2\",\"pages\":\"159-70\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"1978-04-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Investigative & cell pathology\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Investigative & cell pathology","FirstCategoryId":"1085","ListUrlMain":"","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
The mechanisms of human tumour-induced osteolysis and hypercalcaemia.
This is a brief resume of the current knowledge regarding the mechanisms whereby tumours may cause the breakdown or increase the turnover of bone matrix resulting in hypercalcaemia. The precise mechanisms and agents still remain to be ascertained. This would appear to be an investigative area worthy of significant research effort in view of the potential benefit to patient care which may then ensue.
求助内容：
应助结果提醒方式：
京公网安备 11010802042870号
