脑瘫患者的肌肉长度和力量产生-肌腱延长的影响

Pediatric Gait: A New Millennium in Clinical Care and Motion Analysis Technology Pub Date : 2000-07-22 DOI:10.1109/PG.2000.858872

M. Aiona

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引用次数: 2

摘要

肌肉无力是脑瘫患者步态畸形的主要原因。人们担心，延长肌肉可能会加剧这种弱点。采用Eames(1997)所描述的模型测量肌肉长度，在脑瘫患者的12个肢体中，跟腱延长后肌肉长度增加。踝关节的推离发电量增加而不是减少。通过步态分析，适当延长腓肠肌/比目鱼肌可以增加推举力。

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Muscle length and force production in patients with cerebral palsy-the effects of tendon lengthening

Muscle weakness is a major contributor to gait deformity in patients with cerebral palsy. Concern has been raised that lengthening muscles could exacerbate this weakness. Using the model described by Eames (1997) to measure muscle length, increase in muscle length was seen after Achilles tendon lengthening in 12 limbs of patients with cerebral palsy. Push-off power generation at the ankle increased rather than decreased. Appropriate lengthening of the gastrocnemius/soleus muscle can result in an increase in pushoff power as measured by gait analysis.

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Pediatric Gait: A New Millennium in Clinical Care and Motion Analysis Technology

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