{"title":"10. 致癌假说——返祖理论","authors":"Lill-ToveRasmussen Busund","doi":"10.18261/9788215041193-2020-10","DOIUrl":null,"url":null,"abstract":"A deep comprehension of what cancer is as a biological phenomenon is lacking. Several theories have been proposed and many of them do not necessarily contradict each other. One of the theories is the intriguing hypothesis that a cancer cell may be triggered by mutations, but is basically a self-activated throwback to an ancestral cell phenotype running its ancient core functionality by preserving its vital functions, such as survival and uncontrolled proliferation.","PeriodicalId":386489,"journal":{"name":"Advancing Systems Epidemiology in Cancer","volume":"5 1","pages":"0"},"PeriodicalIF":0.0000,"publicationDate":"2020-12-08","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"3","resultStr":"{\"title\":\"10. Hypotheses of Carcinogenesis—The AtavisticTheory\",\"authors\":\"Lill-ToveRasmussen Busund\",\"doi\":\"10.18261/9788215041193-2020-10\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"A deep comprehension of what cancer is as a biological phenomenon is lacking. Several theories have been proposed and many of them do not necessarily contradict each other. One of the theories is the intriguing hypothesis that a cancer cell may be triggered by mutations, but is basically a self-activated throwback to an ancestral cell phenotype running its ancient core functionality by preserving its vital functions, such as survival and uncontrolled proliferation.\",\"PeriodicalId\":386489,\"journal\":{\"name\":\"Advancing Systems Epidemiology in Cancer\",\"volume\":\"5 1\",\"pages\":\"0\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2020-12-08\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"3\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Advancing Systems Epidemiology in Cancer\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.18261/9788215041193-2020-10\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Advancing Systems Epidemiology in Cancer","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.18261/9788215041193-2020-10","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
10. Hypotheses of Carcinogenesis—The AtavisticTheory
A deep comprehension of what cancer is as a biological phenomenon is lacking. Several theories have been proposed and many of them do not necessarily contradict each other. One of the theories is the intriguing hypothesis that a cancer cell may be triggered by mutations, but is basically a self-activated throwback to an ancestral cell phenotype running its ancient core functionality by preserving its vital functions, such as survival and uncontrolled proliferation.