非二尖瓣心血管手术急性左心室流出道阻塞:一个病例系列分析

L. Krichevskiy
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摘要

目的:分析主要非二尖瓣心血管手术围手术期左室流出道梗阻的临床表现及处理方法。设计。案例系列分析。方法与结果患者13例(男10例,女3例),年龄64岁(56;74)纳入了2006年5月至2018年5月期间非二尖瓣心血管手术期间/之后急性出现左室流出道梗阻的患者。方法:行冠状动脉旁路移植术11例,主动脉瓣置换术1例,腹主动脉膜切除术(DeBakey型主动脉夹层,急性下肢缺血)1例。左心室流出道梗阻伴二尖瓣前小叶收缩前移占围术期超声心动图检查总数的0.9%。本文描述了其临床病程的三种变体:(1)通过特定治疗恢复心内和全身血流动力学(大多数病例);(2)完全抵抗治疗,持续收缩前运动;(3)收缩期前运动终止是治疗的结果,但低心输出量综合征却矛盾地持续存在。结论:早期发现急性左室流出道梗阻需要医师的警惕和超声心动图监测。它的发展可以作为低心输出量综合征的原因之一的极硬同心性左心室肥厚的标志。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Acute Left Ventricular Outflow Tract Obstruction in Non - Mitral Cardiovascular Surgery: A Case Series Analysis
Objective: We aimed to analyze the clinical signs of left ventricular outflow tract obstruction and its management in the perioperative period of major non-mitral cardiovascular procedures. Design. Case series analysis. Methods and Results Thirteen (10 males, 3 females) patients aged 64 (56; 74) y.o. with acutely emerged left ventricular outflow tract obstruction during/after non-mitral cardiovascular procedure between May 2006 and May 2018 were included. Methods: The procedures were as follows: coronary artery bypass grafting – n=11, aortic valve replacement – n=1, abdominal aortic membrane resection (aortic dissection DeBakey type I, acute legs ischemia) – n=1. Left ventricular outflow tract obstruction with systolic anterior motion of anterior leaflet of mitral valve was detected in 0.9% of the total number of perioperative echocardiography examinations. Three variants of its clinical course were described: (1) intracardiac and systemic hemodynamics recovery with a specific therapy (most cases); (2) full resistance to therapy with sustainable systolic anterior motion persistence; (3) termination of systolic anterior motion as a result of the therapy, but the paradoxical persistence of low cardiac output syndrome. Conclusion: Practitioners’ vigilance and Echocardiographic monitoring are needed for early detection of acute left ventricular outflow tract obstruction. Its development can be a marker of the extremely hard concentric left ventricular hypertrophy as a cause of the low cardiac output syndrome.
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