血管紧张素转换酶(ACE)插入/缺失(I/D)多态性在不孕症和相关妊娠并发症中作为凝血、纤溶和RAAS途径的联合调节因子

IF 2.1 4区 医学 Q3 PERIPHERAL VASCULAR DISEASE
Sunil Thakur, Vaishnavi Sharma, Dipneet Kaur, Pulakes Purkait
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引用次数: 3

摘要

尽管辅助生殖技术有所增加,但不孕症和妊娠并发症的高发率是全世界不孕症专家关注的主要问题。不孕症可能归因于妊娠并发症,如血栓症、先兆子痫和纤维蛋白诱导的复发性妊娠丢失(RPL)。肾素-血管紧张素-醛固酮系统(RAAS)通过纤溶途径直接或间接导致子痫前期和血栓形成,最终导致RPL或不孕症。这种相互作用的潜在机制尚不清楚。本综述旨在证明RAAS和纤溶途径在妊娠并发症中的作用和相互作用。这种相互作用如何引起妊娠并发症,并最终导致不孕,也讨论了根据目前的证据。本研究还提出了在血栓形成、子痫前期和RPL中RAAS和纤溶过程相关的共同标志物。这些途径的共同联系是ACE基因I/D多态性。除ACE外,PAI-1、VIIa、XIIa、AT1R、AT1AA和TF是常见的分子,可以描述妊娠并发症和不孕的潜在原因。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Angiotensin-Converting Enzyme (ACE) Insertion/Deletion (I/D) Polymorphism as a Conjoint Regulator of Coagulation, Fibrinolytic, and RAAS Pathway in Infertility and Associated Pregnancy Complications.

Angiotensin-Converting Enzyme (ACE) Insertion/Deletion (I/D) Polymorphism as a Conjoint Regulator of Coagulation, Fibrinolytic, and RAAS Pathway in Infertility and Associated Pregnancy Complications.

Angiotensin-Converting Enzyme (ACE) Insertion/Deletion (I/D) Polymorphism as a Conjoint Regulator of Coagulation, Fibrinolytic, and RAAS Pathway in Infertility and Associated Pregnancy Complications.

Angiotensin-Converting Enzyme (ACE) Insertion/Deletion (I/D) Polymorphism as a Conjoint Regulator of Coagulation, Fibrinolytic, and RAAS Pathway in Infertility and Associated Pregnancy Complications.

Despite the increase in assisted reproductive technologies, the high rates of infertility and pregnancy complications are a major concern to infertility specialists worldwide. Infertility may be attributed to pregnancy complications like thrombophilia, preeclampsia and fibrin-induced recurrent pregnancy loss (RPL). Renin-angiotensin-aldosterone system (RAAS) directly or indirectly causes preeclampsia and thrombophilia through the fibrinolytic pathway that ultimately leads to RPL or infertility. The underlying mechanisms of this interaction are still unclear. The present comprehensive review is intended to demonstrate the role and interaction of RAAS and fibrinolytic pathways in pregnancy complications. How this interaction can induce pregnancy complications, and ultimately infertility, is also discussed in the light of current evidence. This study also presents common markers that link RAAS and fibrinolytic processes in developing thrombophilia, preeclampsia and RPL. The common link in these pathways is ACE gene I/D polymorphism. Apart from ACE, PAI-1, VIIa, XIIa, AT1R, AT1AA, and TF are common molecules that can delineate the underlying causes of pregnancy complications and infertility.

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来源期刊
CiteScore
6.20
自引率
0.00%
发文量
16
审稿时长
6-12 weeks
期刊介绍: JRAAS is a peer-reviewed, open access journal, serving as a resource for biomedical professionals, primarily with an active interest in the renin-angiotensin-aldosterone system in humans and other mammals. It publishes original research and reviews on the normal and abnormal function of this system and its pharmacology and therapeutics, mostly in a cardiovascular context but including research in all areas where this system is present, including the brain, lungs and gastro-intestinal tract.
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