停药:中药唐志清介导的NRF2降低糖尿病相关认知障碍HT22小鼠自噬依赖性铁下垂和减轻神经元损伤的实验研究

IF 2 4区 医学 Q3 ENDOCRINOLOGY & METABOLISM
Lingyan Qiu, Kai Chen, Xu Wang, Yun Zhao
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引用次数: 1

摘要

应《内分泌、代谢和免疫疾病药物靶点》杂志作者的要求,该文章已被撤回。Bentham Science对由此造成的不便向该杂志读者道歉。Bentham关于文章撤回的编辑政策可以在https://benthamscience.com/editorial-policies-main.php.Bentham科学免责声明:发表的一个条件是,提交给本杂志的手稿没有发表,也不会同时提交或在其他地方发表。此外,必须报告在其他地方发布的任何数据、插图、结构或表格,并获得复制的版权许可。严禁抄袭,通过提交文章发表,作者同意,如果发现抄袭或捏造信息,出版商有权对作者采取适当行动。通过提交手稿,作者同意,如果文章被接受发表,他们文章的版权将转移给出版商。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Withdrawn: Experimental Study on NRF2 Mediated by Chinese Medicine Tangzhiqing to Reduce Autophagy-dependent Ferroptosis and Alleviate Neuron Damage in HT22 Mice with Diabetes-related Cognitive Disorder

The article has been withdrawn at the request of the authors of the journal Endocrine, Metabolic & Immune Disorders-Drug Targets.

Bentham Science apologizes to the readers of the journal for any inconvenience this may have caused.

The Bentham Editorial Policy on Article Withdrawal can be found at https://benthamscience.com/editorial-policies-main.php.

Bentham science disclaimer: It is a condition of publication that manuscripts submitted to this journal have not been published and will not be simultane-ously submitted or published elsewhere. Furthermore, any data, illustration, structure or table that has been published elsewhere must be reported, and copyright permission for reproduction must be obtained. Plagiarism is strictly forbidden, and by submit-ting the article for publication the authors agree that the publishers have the legal right to take appropriate action against the authors, if plagiarism or fabricated information is discovered. By submitting a manuscript the authors agree that the copyright of their article is transferred to the publishers if and when the article is accepted for publication.

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来源期刊
Endocrine, metabolic & immune disorders drug targets
Endocrine, metabolic & immune disorders drug targets ENDOCRINOLOGY & METABOLISMIMMUNOLOGY-IMMUNOLOGY
CiteScore
4.60
自引率
5.30%
发文量
217
期刊介绍: Aims & Scope This journal is devoted to timely reviews and original articles of experimental and clinical studies in the field of endocrine, metabolic, and immune disorders. Specific emphasis is placed on humoral and cellular targets for natural, synthetic, and genetically engineered drugs that enhance or impair endocrine, metabolic, and immune parameters and functions. Moreover, the topics related to effects of food components and/or nutraceuticals on the endocrine-metabolic-immune axis and on microbioma composition are welcome.
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