Critical Hypophosphatemia in a Special Operations Combat Dive Candidate: A Case Report.

Q3 Medicine
Gerrit Davis, Jeremy Czarnik, Joshua D Evans, Owen L McGrane
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引用次数: 0

Abstract

In contrast to shallow water (hypoxic) blackout and swimming-induced pulmonary edema (SIPE), acute electrolyte disturbance secondary to acute respiratory alkalosis is not considered a common Combat Swimmer injury but has the potential to be life-threatening. We present the case of a 28-year-old Special Operations Dive Candidate who presented to the Emergency Department after a near-drowning incident with altered mental status, generalized weakness, respiratory distress, and tetany. He was found to have severe symptomatic hypophosphatemia (1.00mg/dL) and mild hypocalcemia secondary to intentional hyperventilation between subsurface "cross-overs," causing subsequent acute respiratory alkalosis. This is a unique presentation of a common electrolyte abnormality in a highly specialized population that is self-limiting when caused by acute respiratory alkalosis but poses a significant danger to Combat Swimmers if rescue personnel are not able to respond quickly.

特种作战潜水候选者中的严重低磷血症:一例报告。
与浅水(缺氧)昏迷和游泳引起的肺水肿(SIPE)不同,急性呼吸性碱中毒继发的急性电解质紊乱不被认为是常见的战斗游泳损伤,但有可能危及生命。我们报告了一名28岁的特殊行动潜水候选人,他在溺水事件后出现精神状态改变,全身无力,呼吸窘迫和抽搐。他被发现有严重的症状性低磷血症(1.00mg/dL)和轻度低钙血症,继发于地表下“交叉”间故意过度通气,导致随后的急性呼吸性碱中毒。这是在高度专业化的人群中常见的电解质异常的独特表现,当由急性呼吸性碱中毒引起时,这种异常是自限性的,但如果救援人员不能迅速反应,则对战斗游泳者构成重大危险。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
CiteScore
1.30
自引率
0.00%
发文量
91
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