SARS-CoV-2 infection presenting as sustained atrial flutter and advanced ventricular dysfunction

J. Su, M. Weisert, M. Silka, Y. Bar-Cohen, J. Menteer
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引用次数: 1

Abstract

Viral infections may affect the cardiovascular system in various ways. A number of viruses are known to cause myocarditis and pericarditis, which may result in severe impairment of ventricular function leading to decompensated heart failure [1]. Viral infections may also cause systemic inflammatory response syndrome and septic shock from exaggerated immune inflammatory response [2]. Certain viruses have also been linked to development of cardiac arrhythmias [3,4]. In these cases, evidence of myocardial inflammation is often found. However, arrhythmias are sometimes the only indicator of cardiac involvement in viral infections. Additionally, myocardial failure of any cause can raise atrial and ventricular filling pressures, resulting in increased myocardial fiber stress, potentially inducing arrhythmias or triggering arrhythmias.
SARS-CoV-2感染表现为持续心房扑动和晚期心室功能障碍
病毒感染可以通过多种方式影响心血管系统。已知多种病毒可引起心肌炎和心包炎,可导致严重的心室功能损害,导致失代偿性心力衰竭[1]。病毒感染还可因免疫炎症反应过度而引起全身炎症反应综合征和感染性休克[2]。某些病毒也与心律失常的发生有关[3,4]。在这些病例中,经常发现心肌炎症的证据。然而,心律失常有时是病毒感染中心脏受累的唯一指标。此外,任何原因的心肌衰竭均可提高心房和心室充盈压力,导致心肌纤维应激增加,可能诱发或触发心律失常。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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