Plasma Fibrinogen Concentrations in Patients with Solid Tumors and Therapeutic Improvements by Combining Anticoagulants or FibrinolyticalAgents

D. Lu, Tingren Lu, Chen Xl, B. Xu, J. Ding
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引用次数: 6

Abstract

Many of patient’s deaths with solid tumors are caused by altered pathogenesis coagulation cascade components and processes in clinics. Therapeutic actions for inhibiting cancer coagulation complications in clinics are indispensable part of modern cancer therapies. This editorial discusses the relationship between plasma/solid tumor fibrinogen levels and mechanisms of action by anticoagulants and fibrinolytic agents. Revisit old theory with latest hypothetic and therapeutic options. Many updating clinical information between fibrinogen malformation and its specific therapy are highlighted herein.
实体瘤患者血浆纤维蛋白原浓度与抗凝或纤溶药物联合治疗的改善
临床上许多实体瘤患者的死亡是由于发病机制、凝血级联成分和过程的改变引起的。临床上抑制肿瘤凝血并发症的治疗措施是现代癌症治疗不可缺少的一部分。这篇社论讨论了血浆/实体肿瘤纤维蛋白原水平与抗凝剂和纤维蛋白溶解剂的作用机制之间的关系。用最新的假设和治疗方案重温旧理论。许多最新的临床信息之间的纤维蛋白原畸形和它的具体治疗强调在这里。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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