A. Huacuja, Dafne Arellano Montalvo, A. A. Montalvo
{"title":"Photo Skin Rejuvenation with IPL","authors":"A. Huacuja, Dafne Arellano Montalvo, A. A. Montalvo","doi":"10.15226/2378-1726/8/4/001146","DOIUrl":null,"url":null,"abstract":"Photo aging is becoming increasingly interesting, as it causes, among other alterations, the appearance of wrinkles, pigmentation disorders and loss of normal skin tone. Exposure to sunlight is responsible for 80 to 90% of facial aging. Signs of photoaged skin include changes in skin color, dyschromia’s, lentigos, gradual appearance of telangiectasias (dilation of small blood vessels), as well as changes in skin texture such as loss of smoothness, keratotic appearance, early development and deepening of wrinkles.The aging of our skin is not only the result of the passage of time. It is also strongly influenced by external factors, such as weather conditions. The skin is made up of three layers: the epidermis, or outer layer, the dermis, or middle layer, and the subcutaneous tissue, or deep layer. The dermis contains collagen, elastin, and other fibers that make up the skin’s structure. These elements give the skin a smooth and youthful appearance, and these are those damaged by Ultraviolet Radiation (UVR). UVRs are made up of two different types of waves, UVA and UVB. When UV rays hit the skin, the melanocytes struggle to produce melanin to deposit in the epidermis. This is the process that gives you a tan, which is actually a defense reaction against radiation so that it does not penetrate the skin. UVB rays are shorter than UVA rays and are the main cause of sunburn. UVA rays are responsible for much of the damage we associate with photoaging. UVA rays penetrate deep into the dermis, where damage to collagen fibers occurs. This damage causes an abnormal increase in elastin production.","PeriodicalId":15481,"journal":{"name":"Journal of Clinical Research in Dermatology","volume":"1 1","pages":""},"PeriodicalIF":0.0000,"publicationDate":"2021-08-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"Journal of Clinical Research in Dermatology","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.15226/2378-1726/8/4/001146","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
引用次数: 0
Abstract
Photo aging is becoming increasingly interesting, as it causes, among other alterations, the appearance of wrinkles, pigmentation disorders and loss of normal skin tone. Exposure to sunlight is responsible for 80 to 90% of facial aging. Signs of photoaged skin include changes in skin color, dyschromia’s, lentigos, gradual appearance of telangiectasias (dilation of small blood vessels), as well as changes in skin texture such as loss of smoothness, keratotic appearance, early development and deepening of wrinkles.The aging of our skin is not only the result of the passage of time. It is also strongly influenced by external factors, such as weather conditions. The skin is made up of three layers: the epidermis, or outer layer, the dermis, or middle layer, and the subcutaneous tissue, or deep layer. The dermis contains collagen, elastin, and other fibers that make up the skin’s structure. These elements give the skin a smooth and youthful appearance, and these are those damaged by Ultraviolet Radiation (UVR). UVRs are made up of two different types of waves, UVA and UVB. When UV rays hit the skin, the melanocytes struggle to produce melanin to deposit in the epidermis. This is the process that gives you a tan, which is actually a defense reaction against radiation so that it does not penetrate the skin. UVB rays are shorter than UVA rays and are the main cause of sunburn. UVA rays are responsible for much of the damage we associate with photoaging. UVA rays penetrate deep into the dermis, where damage to collagen fibers occurs. This damage causes an abnormal increase in elastin production.