[Secondary open-angle glaucoma: uveitic secondary glaucoma, steroid-induced glaucoma, posttraumatic and postoperative glaucoma, tumor-related glaucoma and glaucoma due to elevated episcleral venous pressure].

Der Ophthalmologe Pub Date : 2022-05-01 Epub Date: 2022-04-26 DOI:10.1007/s00347-022-01630-6
Roman Greslechner, Horst Helbig, Detlev Spiegel
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引用次数: 0

Abstract

Secondary open-angle glaucomas are a heterogeneous group of diseases in which a variety of pathophysiological mechanisms result in an elevation of intraocular pressure (IOP). In contrast to primary open-angle glaucoma in many cases besides IOP reduction a causal treatment is possible. This article is the second part of a review of the more frequently encountered forms of secondary open-angle glaucoma. Uveitic glaucoma, steroid-induced glaucoma, posttraumatic and postoperative glaucoma, glaucoma due to intraocular tumors and caused by elevated episcleral venous pressure are covered. The underlying pathophysiological mechanisms, characteristic clinical findings and treatment options are discussed.

【继发性开角型青光眼:青光眼继发性青光眼、类固醇性青光眼、创伤后及术后青光眼、肿瘤相关性青光眼和锁膜外静脉压升高所致青光眼】。
继发性开角型青光眼是一种异质性疾病,多种病理生理机制可导致眼压升高。与原发性开角型青光眼相比,在许多病例中,除了IOP降低外,因果治疗是可能的。本文是对继发性开角型青光眼的第二部分综述。包括葡萄膜性青光眼、类固醇性青光眼、创伤后和术后青光眼、眼内肿瘤和锁骨外静脉压升高引起的青光眼。讨论了潜在的病理生理机制、临床表现和治疗方案。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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