A

Hans Jörg Schrötter
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Abstract

peritoneal lavage was carried out, employing a peritoneal dialysis catheter, inserted through a small sub-umbilical incision in skin and linea alba. Five hundred ml normal saline were run in and returned clear. The catheter was removed and the skin closed with a single nylon stitch. Erect chest and supine abdominal films were normal. The patient was managed conservatively with intravenous fluids and regular careful observation. Two h after presentation, while her general condition was unchanged, palpation revealed very obvious surgical emphysema in the lower abdominal wall. On a second erect chest X-ray, gas was clearly demonstrated under both hemi-diaphragms and laparotomy was, therefore, undertaken. There was a considerable amount of bilestained fluid and gas in the retroperitoneal tissues around the duodenum, but only a small amount of free fluid in the peritoneal cavity. The duodenum was mobilised to display a 2 cm tear on the posterior surface of the junction of the second and third parts which was repaired. The patient made an uneventful post-operative recovery. The detection of surgical emphysema in the anterior abdominal wall clearly heightened suspicion of major intra-abdominal injury. The gas must have come from a perforated or leaking viscus. The possibility of iatrogenic perforation by the dialysis catheter was considered, but seemed unlikely as there was no faecal or bile-staining of the returning irrigant. Since initial X-rays were normal, slow leakage of gas from the retroduodenal tissues into the lesser sac and eventually into the general peritoneal cavity seems a likely sequence of events. In the supine patient, gas could rise to be expelled by contraction of the abdominal musculature through the small slit in the linea and into the subcutaneous tissues. This mechanism produced an unexpected but important clinical finding following initially negative peritoneal lavage which lead to the early detection of traumatic duodenal rupture, an injury in which diagnosis and treatment are commonly delayed for over 24 hours, and complications and mortality are correspondingly high.
一个
腹腔灌洗,采用腹膜透析导管,通过皮肤和白线的小脐下切口插入。注入500毫升生理盐水,返回清澈。取下导管,用尼龙针缝合皮肤。直立胸片和仰卧腹部片正常。对患者进行了保守的静脉输液和定期仔细观察。就诊后2小时,患者一般情况无变化,触诊发现下腹壁有非常明显的外科肺气肿。在第二次直立胸片上,在半膈下明显可见气体,因此进行了剖腹手术。十二指肠周围腹膜后组织内可见大量胆汁染色的液体和气体,腹腔内仅有少量游离液体。十二指肠活动显示第二部分和第三部分连接处的后表面有2厘米的撕裂,该撕裂已修复。病人术后恢复顺利。在前腹壁发现手术肺气肿明显增加了对腹内损伤的怀疑。气体一定来自穿孔或泄漏的内脏。考虑了透析导管医源性穿孔的可能性,但似乎不太可能,因为返回的冲洗液没有粪便或胆汁染色。由于最初的x光片显示正常,气体从十二指肠后组织缓慢渗漏到小囊并最终进入腹膜腔似乎是一个可能的事件序列。仰卧位病人,腹部肌肉收缩,气体上升,经气管小缝排出,进入皮下组织。这种机制在最初的腹膜灌洗阴性后产生了一个意想不到但重要的临床发现,导致早期发现外伤性十二指肠破裂,这种损伤的诊断和治疗通常延迟超过24小时,并发症和死亡率相应很高。
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