Corneal hydrops induced by Bell's paralysis in a case of corneal ectasia

L. Aslan, M. Aslankurt, G. Özdemir, Bedia Sahin, A. Comez
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Abstract

An 18-year-old male patient presented with suddenly decreased vision, itching, corneal edema and an inability to close the left eye. He had left Bell’s paralysis for two weeks and used high diopter glasses for five years. The best corrected visual acuity was 0.4 in his right eye and counting fingers in the left eye. Biomicroscopic examination revealed thinning and steepening of the cornea in the right eye and anterior protrusion of the cornea, stromal edema and punctate disruption of the epithelium in the left eye. Topographic image of the right eye was consistent with keratoconus. Six months later, stromal edema gradually regressed and a corneal scar ensued. This case presentation emphasizes that Bell’s palsy may induce disease progression in a patient with preexisting corneal ectasia and results in corneal hydrops.
角膜扩张症贝尔麻痹致角膜积液1例
一位18岁的男性患者表现为视力突然下降,瘙痒,角膜水肿和无法闭上左眼。他已经离开贝尔麻痹症两周了,戴了五年的高屈光度眼镜。最佳矫正视力为右眼0.4,左眼数指。生物显微镜检查显示右眼角膜变薄变陡,角膜前突,角膜间质水肿,左眼上皮点状破坏。右眼地形图与圆锥角膜一致。6个月后,间质水肿逐渐消退,角膜瘢痕出现。本病例报告强调,贝尔氏麻痹可能会导致先前存在的角膜扩张患者的疾病进展并导致角膜积液。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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