Abstract Book of 7th Brain Research School (2022-BRS), 27 June and 3 July 2022, Isparta, Turkey (http://2022.brs.org.tr/)

Q4 Biochemistry, Genetics and Molecular Biology

Journal of Cellular Neuroscience and Oxidative Stress Pub Date : 2022-07-14 DOI:10.37212/jcnos.1143834

Anonymes ABSTRACT BOOK

{"title":"Abstract Book of 7th Brain Research School (2022-BRS), 27 June and 3 July 2022, Isparta, Turkey (http://2022.brs.org.tr/)","authors":"Anonymes ABSTRACT BOOK","doi":"10.37212/jcnos.1143834","DOIUrl":null,"url":null,"abstract":"7th Brain Research School (2022-BRS) was performed in Isparta, Turkey between 27 June and 3 July 2022 (http://2022.brs.org.tr/). There were abstracts of 8 speakers, 11 oral presentations, and 3 poster presentations in the 2022-BRS.\nTitles of speakers;\n1- Mustafa NAZIROĞLU. Fluorescent Ca2+ stains for imaging the mice microglia.\n2- Beatrice Mihaela RADU. Low-energy accelerated protons irradiation inhibits DNA repair and diminishes cell proliferation, migration and calcium signaling in brain microvascular endothelial cells.\n3- Nashat ABUMARIA. Behavioral assays and animal models of psychiatric disorders.\n4- Ferah YILDIRIM. Mapping genome-wide DNA methylation changes in alcohol use disorder (No abstrcat).\n5- Marco CANEPARI. Principles of Ca2+ imaging using low-affinity indicators.\n6- Denis ROUSSEAU. Western blot analyses in the mitochondria.\n7- Xinhua SHU. Protection of p-Coumaric acid against depression and memory impairment via inhibition of neuroinflammation\n8- Simon HEBEISEN. State dependent block of voltage gated sodium and calcium channels as modern treatment for\nepilepsy.\n\nTitles of oral presentations (OPs);\nOP1. Bünyamin AYDIN. Diabetic neuropathic pain and TRPM2 Channel: Focus on selenium.\nOP2. Agomelatine attenuates calcium signaling and apoptosis via the inhibition of TRPV1\nchannel in the hippocampal neurons of rats with chronic mild stress depression model.\nOP3. Hatice DALDAL. An interaction between cisplatin-induced optic nerve injury and TRPM2 channel.\nOP4. Kemal ERTİLAV. Recent developments on the traumatic brain injury models in the experimental animals.\nOP5. Kaan ERBAKAN. Involvement of TRPV1 channel in the etiology of epilepsy.\nOP6. Ramazan ÇINAR. Does GSH depletion induce TRPM2 activation in neuronal cells?\nOP7. Esin AKBAY ÇETİN. TRP ion channels and approaches in COVID-19.\nOP8. Gülin ÖZDAMAR ÜNAL. Early life stress and neuroinflammation.\nOP9. Adem AHLATCI. Investigation of TRPM2 cation channel activation in PTZ-induced SH-SY5Y cells by patch-clamp technique: Regulatory role of valproic acid.\nOP10. Yener YAZĞAN. Orthodontic teeth movement-induced pain and TRPV1 channel.\nOP11. Evaluation of the role of Radish (Raphanus sativus) extract in movement tests in MPTP induced experimental Parkinson's model in Balb/C mice.\n\nTitles of poster presentations (P);\nP1- Esin AKBAY ÇETİN. Probable pathways of SARS-CoV-2 to central nervous system.\nP2. Yusuf DAL. Low molecular weight heparin treatment reduced apoptosis, oxidative stress, and calcium signaling in the thrombocytes of patients with recurrent pregnancy loss and thrombophilia: Involvements of TRPM2 and TRPV1 channels.\nP3. Ferruh KARAMANGİL. Investigation of frequency and diversity of experimental animal models of schizophrenia.","PeriodicalId":37782,"journal":{"name":"Journal of Cellular Neuroscience and Oxidative Stress","volume":" ","pages":""},"PeriodicalIF":0.0000,"publicationDate":"2022-07-14","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"Journal of Cellular Neuroscience and Oxidative Stress","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.37212/jcnos.1143834","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q4","JCRName":"Biochemistry, Genetics and Molecular Biology","Score":null,"Total":0}

引用次数: 0

Abstract

7th Brain Research School (2022-BRS) was performed in Isparta, Turkey between 27 June and 3 July 2022 (http://2022.brs.org.tr/). There were abstracts of 8 speakers, 11 oral presentations, and 3 poster presentations in the 2022-BRS. Titles of speakers; 1- Mustafa NAZIROĞLU. Fluorescent Ca2+ stains for imaging the mice microglia. 2- Beatrice Mihaela RADU. Low-energy accelerated protons irradiation inhibits DNA repair and diminishes cell proliferation, migration and calcium signaling in brain microvascular endothelial cells. 3- Nashat ABUMARIA. Behavioral assays and animal models of psychiatric disorders. 4- Ferah YILDIRIM. Mapping genome-wide DNA methylation changes in alcohol use disorder (No abstrcat). 5- Marco CANEPARI. Principles of Ca2+ imaging using low-affinity indicators. 6- Denis ROUSSEAU. Western blot analyses in the mitochondria. 7- Xinhua SHU. Protection of p-Coumaric acid against depression and memory impairment via inhibition of neuroinflammation 8- Simon HEBEISEN. State dependent block of voltage gated sodium and calcium channels as modern treatment for epilepsy. Titles of oral presentations (OPs); OP1. Bünyamin AYDIN. Diabetic neuropathic pain and TRPM2 Channel: Focus on selenium. OP2. Agomelatine attenuates calcium signaling and apoptosis via the inhibition of TRPV1 channel in the hippocampal neurons of rats with chronic mild stress depression model. OP3. Hatice DALDAL. An interaction between cisplatin-induced optic nerve injury and TRPM2 channel. OP4. Kemal ERTİLAV. Recent developments on the traumatic brain injury models in the experimental animals. OP5. Kaan ERBAKAN. Involvement of TRPV1 channel in the etiology of epilepsy. OP6. Ramazan ÇINAR. Does GSH depletion induce TRPM2 activation in neuronal cells? OP7. Esin AKBAY ÇETİN. TRP ion channels and approaches in COVID-19. OP8. Gülin ÖZDAMAR ÜNAL. Early life stress and neuroinflammation. OP9. Adem AHLATCI. Investigation of TRPM2 cation channel activation in PTZ-induced SH-SY5Y cells by patch-clamp technique: Regulatory role of valproic acid. OP10. Yener YAZĞAN. Orthodontic teeth movement-induced pain and TRPV1 channel. OP11. Evaluation of the role of Radish (Raphanus sativus) extract in movement tests in MPTP induced experimental Parkinson's model in Balb/C mice. Titles of poster presentations (P); P1- Esin AKBAY ÇETİN. Probable pathways of SARS-CoV-2 to central nervous system. P2. Yusuf DAL. Low molecular weight heparin treatment reduced apoptosis, oxidative stress, and calcium signaling in the thrombocytes of patients with recurrent pregnancy loss and thrombophilia: Involvements of TRPM2 and TRPV1 channels. P3. Ferruh KARAMANGİL. Investigation of frequency and diversity of experimental animal models of schizophrenia.

查看原文本刊更多论文

第七脑研究学院(2022- brs)摘要，2022年6月27日和7月3日，土耳其伊斯帕塔(http://2022.brs.org.tr/)

第七脑研究学校(2022- brs)于2022年6月27日至7月3日在土耳其伊斯帕塔进行(http://2022.brs.org.tr/)。在2022-BRS中，有8位演讲者的摘要，11个口头报告和3个海报报告。演讲者的头衔;1- Mustafa NAZIROĞLU。荧光Ca2+染色用于小鼠小胶质细胞成像。2- Beatrice Mihaela RADU。低能加速质子辐照抑制脑微血管内皮细胞DNA修复，减少细胞增殖、迁移和钙信号。3-纳什特·阿布马里亚。精神疾病的行为分析和动物模型。费拉·耶尔德琳。绘制全基因组DNA甲基化在酒精使用障碍中的变化(无摘要)。马尔科·卡内帕里。使用低亲和力指标的Ca2+成像原理。6-丹尼斯·卢梭线粒体的Western blot分析。——舒新华。对香豆酸通过抑制神经炎症对抑郁和记忆障碍的保护作用[j]电压门控钠钙通道的状态依赖性阻断作为癫痫的现代治疗方法。口头报告的题目;Bunyamin艾登。糖尿病神经性疼痛与TRPM2通道:关注硒和op2。阿戈美拉汀通过抑制慢性轻度应激性抑郁模型大鼠海马神经元trpv1通道减弱钙信号和细胞凋亡。Hatice DALDAL。顺铂诱导的视神经损伤与TRPM2通道的相互作用。凯末尔ERTİ盥洗室。实验动物创伤性脑损伤模型的研究进展[j]。Kaan尔巴坎。TRPV1通道在癫痫病因学中的作用。斋月CINAR。GSH耗竭是否会诱导神经元细胞中TRPM2的激活?Esin AKBAY ÇETİN。covid -19中的TRP离子通道和途径g林ÖZDAMAR ÜNAL。早期生活压力和神经炎症。亚当AHLATCI。膜片钳技术对ptz诱导SH-SY5Y细胞TRPM2阳离子通道激活的研究:丙戊酸的调节作用[j]。日元YAZĞ。正畸牙齿运动引起的疼痛与TRPV1通道。萝卜提取物在MPTP诱导的Balb/C小鼠帕金森模型运动试验中的作用评价。海报展示标题(P);P1- Esin AKBAY ÇETİN。SARS-CoV-2进入中枢神经系统的可能途径。优素福木豆。低分子肝素治疗可减少复发性妊娠丢失和血栓患者血栓细胞的凋亡、氧化应激和钙信号:TRPM2和TRPV1通道的参与。Ferruh KARAMANGİL。精神分裂症实验动物模型的频率和多样性调查。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

求助全文

约1分钟内获得全文求助全文

来源期刊

Journal of Cellular Neuroscience and Oxidative Stress Biochemistry, Genetics and Molecular Biology-Biophysics

CiteScore

1.10

自引率

0.00%

发文量

期刊介绍： Journal of Cellular Neuroscience and Oxidative Stress isan online journal that publishes original research articles, reviews and short reviews on themolecular basisofbiophysical,physiological and pharmacological processes thatregulate cellular function, and the control or alteration of these processesby theaction of receptors, neurotransmitters, second messengers, cation, anions,drugsor disease. Areas of particular interest are four topics. They are; 1. Ion Channels (Na+-K+Channels, Cl– channels, Ca2+channels, ADP-Ribose and metabolism of NAD+,Patch-Clamp applications) 2. Oxidative Stress (Antioxidant vitamins, antioxidant enzymes, metabolism of nitric oxide, oxidative stress, biophysics, biochemistry and physiology of free oxygen radicals) 3. Interaction Between Oxidative Stress and Ion Channels in Neuroscience (Effects of the oxidative stress on the activation of the voltage sensitive cation channels, effect of ADP-Ribose and NAD+ on activation of the cation channels which are sensitive to voltage, effect of the oxidative stress on activation of the TRP channels in neurodegenerative diseases such Parkinson’s and Alzheimer’s diseases) 4. Gene and Oxidative Stress (Gene abnormalities. Interaction between gene and free radicals. Gene anomalies and iron. Role of radiation and cancer on gene polymorphism)