Epicardial Pacemaker Lead-induced Cardiac Strangulation Causing Asystole due to Unexpected Myocardial Ischemia during Catheterization: A Case Report

Hiromitsu Shirozu, Y. Ishikawa, Ayako Kuraoka, Y. Kodama, Makoto Nakamura, H. Ushinohama, Koichi Sagawa, Joji Hashimoto, Y. Ando, T. Nakano
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Abstract

A 16-year-old male with complete congenital atrioventricular block since 1 year old was treated by epicardial pacemaker lead implantation. At 9 years old, he was diagnosed with supravalvular pulmonary artery stenosis resulting from epicardial pacemaker leads with cardiac strangulation as the possible cause. Transient ST change was seen only in the exercise tolerance test, with no chest pain, indicating that myocardial ischemia was absent. The deterioration status of supravalvular pulmonary artery stenosis was assessed by catheterization. With his arms raised, he was injected with a small amount of contrast media, suddenly, he lost consciousness. Electrocardiography showed ST segment alterations after frequent premature ventricular contractions, followed by junc-tional bradycardia that rapidly progressed to asystole. Hence, transient ischemia caused by arm raising or frequent premature ventricular contractions and deterioration of the coronary artery compression was suspected. The leads were then removed in an urgent surgery. In conclusion, contrast-enhanced computed tomography should be conducted in patients with cardiac strangulation, and earlier lead exchange should be performed because of the rapid deterioration of myocardial ischemia.
心外膜起搏器铅致心脏绞窄导致心导管置管过程中意外心肌缺血导致心脏停止:1例报告
一名16岁男性,自1岁起完全性先天性房室传导阻滞,接受心外膜起搏器导线植入治疗。9岁时,他被诊断为心外膜起搏器导线引起的瓣上肺动脉狭窄,可能的原因是心脏勒死。仅在运动耐受性试验中观察到短暂的ST段变化,没有胸痛,表明心肌缺血不存在。通过导管插入术评估瓣膜上肺动脉狭窄的恶化情况。他举起双臂,注射了少量的造影剂,突然,他失去了意识。心电图显示,频繁的室性早搏后ST段改变,随后出现快速发展为心搏停止的交界性心动过缓。因此,有人怀疑由抬臂或频繁的室性早搏和冠状动脉压迫恶化引起的短暂性缺血。随后在紧急手术中取出了导线。总之,心脏绞杀患者应进行对比增强计算机断层扫描,并且由于心肌缺血的快速恶化,应尽早进行导线交换。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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