Collagen cross-link breakers: a beginning of a new era in the treatment of cardiovascular changes associated with aging, diabetes, and hypertension.

Dinko Susic, Jasmina Varagic, Jwari Ahn, Edward D Frohlich
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引用次数: 40

Abstract

Aging, diabetes, and hypertension are conditions in which arterial and myocardial stiffness is increased. Increased arterial stiffness is manifested by an increased systolic arterial pressure, pulse pressure and pulse wave velocity, whereas increased myocardial stiffness is manifested by impaired left ventricular diastolic filling. Moreover, increased arterial stiffness increases cardiac workload, further aggravating already existing adverse changes in left ventricular structure and function. Indeed, studies in human beings have clearly shown that increased cardiovascular stiffness is a reliable predictor of cardiovascular morbidity and mortality. Increased cardiovascular stiffness is usually attributed to the development of fibrosis (i.e., accumulation of collagen). It has also been recognized that the increased cardiac and vascular stiffness may be due to increased collagen cross-linking due to the formation of advanced glycosylation end-products (AGEs). In agreement with this notion is the finding that an inhibitor of AGEs formation improves vascular stiffness in diabetic rats. More recently, cross-link breakers have been developed, and the beneficial effects of one such agent (ALT-711) have been shown in experimental and clinical settings. This report briefly summarizes age related changes in cardiovascular structure and function and describes results of experimental and clinical studies involving collagen cross-link breakers.

胶原交联抑制剂:与衰老、糖尿病和高血压相关的心血管变化治疗新时代的开始
衰老、糖尿病和高血压都是动脉和心肌僵硬度增加的情况。动脉僵硬度增加表现为动脉收缩压、脉压和脉波速度增加,而心肌僵硬度增加表现为左心室舒张充盈受损。此外,动脉僵硬度的增加增加了心脏负荷,进一步加重了已经存在的左心室结构和功能的不良变化。事实上,对人类的研究已经清楚地表明,心血管僵硬度的增加是心血管发病率和死亡率的可靠预测指标。心血管僵硬的增加通常归因于纤维化的发展(即胶原蛋白的积累)。人们也认识到,心脏和血管硬度的增加可能是由于晚期糖基化终产物(AGEs)形成导致胶原交联增加。与这一观点一致的是,发现AGEs形成的抑制剂可以改善糖尿病大鼠的血管僵硬。最近,交联抑制剂已被开发出来,其中一种药物(ALT-711)的有益作用已在实验和临床环境中得到证实。本文简要总结了年龄对心血管结构和功能的影响,并介绍了胶原交联抑制剂的实验和临床研究结果。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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