Wnt signaling in biliary development, proliferation, and fibrosis.

IF 2.7
Lipeng Tian, Yichen Wang, Yoon Young Jang
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引用次数: 2

Abstract

Biliary fibrosis is an important pathological indicator of hepatobiliary damage. Cholangiocyte is the key cell type involved in this process. To reveal the pathogenesis of biliary fibrosis, it is essential to understand the normal development as well as the aberrant generation and proliferation of cholangiocytes. Numerous reports suggest that the Wnt signaling pathway is implicated in the physiological and pathological processes of cholangiocyte development and ductular reaction. In this review, we summarize the effects of Wnt pathway in cholangiocyte development from embryonic stem cells, as well as the underlying mechanisms of cholangiocyte responses to adult ductal damage. Wnt signaling pathway is regulated in a step-wise manner during each of the liver differentiation stages from embryonic stem cells to functional mature cholangiocytes. With the modulation of Wnt pathway, cholangiocytes can also be generated from adult liver progenitor cells and mature hepatocytes to repair liver damage. Non-canonical Wnt signaling is triggered in the active ductal cells during biliary fibrosis. Targeted control of the Wnt signaling may hold the great potential to reduce and/or reverse the biliary fibrogenic process.

Wnt信号在胆道发育、增殖和纤维化中的作用。
胆道纤维化是肝胆损害的重要病理指标。胆管细胞是参与这一过程的关键细胞类型。为了揭示胆道纤维化的发病机制,有必要了解胆管细胞的正常发育以及异常的产生和增殖。大量报道表明,Wnt信号通路参与了胆管细胞发育和导管反应的生理和病理过程。本文综述了Wnt通路在胚胎干细胞胆管细胞发育中的作用,以及胆管细胞对成人胆管损伤反应的潜在机制。Wnt信号通路在肝脏从胚胎干细胞向功能成熟的胆管细胞分化的每个阶段都以一种循序渐进的方式受到调节。在Wnt通路的调控下,成人肝祖细胞和成熟肝细胞也可生成胆管细胞,修复肝损伤。在胆道纤维化过程中,活跃的导管细胞会触发非典型Wnt信号。有针对性地控制Wnt信号可能具有减少和/或逆转胆道纤维化过程的巨大潜力。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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