Structural Failure Mechanisms of Common Flexor Tendon Repairs.

Tim Sebastian Peltz, Roger Haddad, Peter James Scougall, Sean Nicklin, Mark Peter Gianoutsos, Rema Oliver, William Robert Walsh
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引用次数: 7

Abstract

Background: This study investigated the exact failure mechanisms of the most commonly used conventional tendon repair techniques. A new method, radiographing repair constructs in antero-posterior and lateral projections before and after tensioning was used. This allowed to precisely analyse failure mechanisms in regards to geometrical changes in all three dimensions. Additionally the biomechanical stability focusing on gapping was tested.

Methods: Sheep fore limb deep flexor tendons were harvested and divided in eight groups of ten tendons. Three common variants of the Kessler repair method and four common 4-strand repair techniques were tested. Additionally a new modification of the Adelaide repair was tested.

Results: Biomechanical testing showed no significant differences in gapping for the three tested 2-strand Kessler repair groups. Once a double Kessler or 4-strand Kessler repair was performed the stability of the repair improved significantly. Further significant improvements in biomechanical stability could be achieved by using cross locks in the repair like in the Adelaide repair method. Qualitative analysis using radiographs showed that all Kessler repair variants unfolded via rotations around the transverse suturing component, no matter which variant was used.

Conclusions: Additional to the commonly described constriction of the repair construct, the rotating deformation is the main reason for repair site gapping in Kessler tendon repair methods. The term "locking" in a Kessler repair is misleading. The cruciate repairs tended to loose grip and drag (cheese-wire) through the tendon and therefore lead to gapping. The most stable repair constructs in all three dimensions were the Adelaide repair and its interlocking modification. This is due to the superior anchoring qualities of its cross locks and three dimensional stability.

屈肌腱修复的结构失效机制。
背景:本研究探讨了最常用的常规肌腱修复技术的确切失效机制。采用了一种新的方法,在张紧前后前后对前后侧位投影进行x线摄影修复。这使得我们可以精确地分析三维几何变化的失效机制。此外,还测试了以间隙为中心的生物力学稳定性。方法:取绵羊前肢深屈肌腱,分为8组,每组10根。测试了Kessler修复方法的三种常见变体和四种常见的四链修复技术。此外,我们还测试了阿德莱德号的新改型。结果:生物力学测试显示,3个2链Kessler修复组的间隙无显著差异。一旦双Kessler或4链Kessler修复进行修复的稳定性显著提高。在阿德莱德修复方法中使用交叉锁可以进一步显著改善生物力学稳定性。x线片定性分析显示,无论使用哪种类型的Kessler修复变体,所有的Kessler修复变体都是通过围绕横向缝合组件旋转展开的。结论:除了常见的修复结构收缩外,旋转变形是凯斯勒肌腱修复方法中修复部位间隙的主要原因。术语“锁定”在凯斯勒修理是误导。十字韧带修复术容易使肌腱松动并拖拉(奶酪丝),从而导致裂口。在所有三个维度中,最稳定的修复结构是阿德莱德修复及其连锁修饰。这是由于其优良的锚固质量的交叉锁和三维稳定性。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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