The role of different subsets of regulatory T cells in immunopathogenesis of rheumatoid arthritis.

Arthritis Pub Date : 2012-01-01 Epub Date: 2012-10-24 DOI:10.1155/2012/805875
Maryam Gol-Ara, Farhad Jadidi-Niaragh, Reza Sadria, Gholamreza Azizi, Abbas Mirshafiey
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引用次数: 78

Abstract

Rheumatoid arthritis (RA) is a common autoimmune disease and a systemic inflammatory disease which is characterized by chronic joint inflammation and variable degrees of bone and cartilage erosion and hyperplasia of synovial tissues. Considering the role of autoreactive T cells (particularly Th1 and Th17 cells) in pathophysiology of RA, it might be assumed that the regulatory T cells (Tregs) will be able to control the initiation and progression of disease. The frequency, function, and properties of various subsets of Tregs including natural Tregs (nTregs), IL-10-producing type 1 Tregs (Tr1 cells), TGF-β-producing Th3 cells, CD8(+) Tregs, and NKT regulatory cells have been investigated in various studies associated with RA and collagen-induced arthritis (CIA) as experimental model of this disease. In this paper, we intend to submit the comprehensive information about the immunobiology of various subsets of Tregs and their roles and function in immunopathophysiology of RA and its animal model, CIA.

调节性T细胞不同亚群在类风湿关节炎免疫发病机制中的作用。
类风湿关节炎(RA)是一种常见的自身免疫性疾病和全身性炎症疾病,其特征是慢性关节炎症和不同程度的骨和软骨侵蚀以及滑膜组织增生。考虑到自身反应性T细胞(特别是Th1和Th17细胞)在RA病理生理中的作用,可以假设调节性T细胞(Tregs)能够控制疾病的发生和进展。各种Tregs亚群的频率、功能和特性,包括天然Tregs (nTregs)、产生il -10的1型Tregs (Tr1细胞)、产生TGF-β的Th3细胞、CD8(+) Tregs和NKT调节细胞,已在各种与RA和胶原诱导关节炎(CIA)相关的研究中作为该疾病的实验模型进行了研究。在本文中,我们打算提交有关Tregs各亚群的免疫生物学及其在RA及其动物模型CIA免疫病理生理中的作用和功能的综合信息。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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