The Mechanisms of Compound 48/80-Induced Superoxide Generation Mediated by A-Kinase in Rat Peritoneal Mast Cells

Biochemical and molecular medicine Pub Date : 1997-06-01 Epub Date: 2002-05-25 DOI:10.1006/bmme.1997.2594
Nobuyuki Fukuishi , Masakiyo Sakaguchi , Shiori Matsuura , Chizuko Nakagawa , Reiko Akagi , Masaaki Akagi
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引用次数: 34

Abstract

This investigation was undertaken to clarify the mechanisms of superoxide anion (O2−) generation in rat peritoneal mast cells. Compound 48/80, a typical histamine liberator mediated by calcium influx, elicited O2−generation from the mast cells in a dose-dependent fashion. It was demonstrated by immunohistochemical study and Western blot analysis that the mast cells contained the 47-kDa phagocyte oxidase (p47phox) protein, which was one cytosolic component of the NADPH oxidase system. Arachidonic acid stimulated O2−generation in the mast cells, but other unsaturated fatty acids had no effect. On the other hand, 48/80-induced O2−generation was inhibited by phospholipase A2 inhibitors, such as arachidonyl trifluoromethyl ketone and manoalide. Forskolin, isoprenaline, and dibutyryl cyclic AMP inhibited the O2−generation, and KT-5720, a cyclic AMP-dependent protein kinase (A-kinase) inhibitor, markedly enhanced the O2−generation. These findings suggest that O2−is generated by a NADPH oxidase-like enzyme system in mast cells and that this enzyme system is activated by arachidonic acid released by cytosolic phospholipase A2. Thus, it is regulated by the cyclic AMP-A kinase system.

a激酶介导的化合物48/80诱导大鼠腹膜肥大细胞产生超氧化物的机制
本研究旨在阐明大鼠腹膜肥大细胞中超氧阴离子(O2−)产生的机制。化合物48/80是一种典型的由钙内流介导的组胺解毒剂,以剂量依赖性的方式诱导肥大细胞产生O2−。免疫组化和Western blot分析表明,肥大细胞含有47-kDa的吞噬细胞氧化酶(p47phox)蛋白,该蛋白是NADPH氧化酶系统的胞质组分之一。花生四烯酸刺激肥大细胞产生O2−,而其他不饱和脂肪酸没有作用。另一方面,48/80诱导的O2 -生成被磷脂酶A2抑制剂(如花生四烯酮三氟甲基酮和manoalide)抑制。Forskolin、异丙肾上腺素和二丁基环AMP均抑制O2 -生成,而环AMP依赖性蛋白激酶(a激酶)抑制剂KT-5720显著增强O2 -生成。这些发现表明,O2−是由肥大细胞中的NADPH氧化酶样酶系统产生的,该酶系统被胞质磷脂酶A2释放的花生四烯酸激活。因此,它受环AMP-A激酶系统的调节。
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