[Monitoring of intracranial pressure].

Cahiers d'anesthesiologie Pub Date : 1996-01-01
F Artru
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Abstract

The use of intraparenchymatous ICP sensor is becoming increasingly popular at the expense of the traditional intraventricular catheter method, in spite of the impossibility, with the former technic, to correct a possible zero drift. The decision to initiate or discontinue ICP monitoring is essentially based upon whether suggestive aspects of raised ICP are or not present on CT-scan. The degree of basal cisterns effacement is particularly informative. The same data from successive CT-scans are used to check the validity of the monitoring. Predefining critical levels of ICP and cerebral perfusion pressure (CPP) allows to establish practical guide-lines for treatment. Cerebral ischemia is considered very likely when ICP rises above 30 mmHg. Regarding CPP, the therapeutical goal is to avoid its reduction under the critical level of 60-80 mmHg. As these thresholds vary with the patients age and the type of lesion, a parallel evaluation of cerebral ischemia by other methods is mandatory. Transcranial doppler allows an easy detection of critical reduction of arterial flow. However, in case of flow hypervelocity, interpretation needs measurement of absolute cerebral blood flow values. Cerebral venous oxygen saturation monitoring, at the level of the jugular golf, shows desaturation episodes indicative of cerebral ischemia. Blood sampling for determination of arterial and jugular venous lactate concentrations allows calculation of the lactate oxygen index, a practical correlate of the degree of cerebral ischemia. ICP measurement alone is of limited value to understand the cerebral hemodynamical and metabolical situation in severe brain injury. Preceding the rise of ICP, there exists a compensation phase during which a progressive decrease of intracranial compliance is the important event. Even more earlier, posttraumatic cellular metabolic dysfunctions are to-day objectives for a neurochemical monitoring. Therefore a special technical and human environment has became mandatory to take a real benefit from ICP monitoring.

[颅内压监测]。
脑实质内ICP传感器的使用越来越受欢迎,其代价是传统的脑室内导管方法,尽管前者技术不可能纠正可能的零漂移。开始或停止ICP监测的决定基本上取决于ct扫描上是否存在ICP升高的提示方面。基底池的消蚀程度尤其能提供信息。来自连续ct扫描的相同数据被用来检查监测的有效性。预先确定ICP和脑灌注压(CPP)的临界水平可以建立实用的治疗指南。当ICP高于30mmhg时,认为很可能是脑缺血。关于CPP,治疗目标是避免其降至60-80 mmHg的临界水平以下。由于这些阈值随患者的年龄和病变类型而变化,因此必须通过其他方法对脑缺血进行平行评估。经颅多普勒可以很容易地检测到动脉血流的临界减少。然而,在血流超高速的情况下,解释需要测量绝对脑血流量值。脑静脉血氧饱和度监测显示,颈静脉水平的血氧饱和度下降表明脑缺血。血液取样测定动脉和颈静脉乳酸浓度可以计算乳酸氧指数,这是脑缺血程度的实际相关指标。单纯颅内压测量对了解重型颅脑损伤患者脑血流动力学和代谢情况的价值有限。在颅内压升高之前,存在一个代偿期,在此期间,颅内顺应性的逐渐降低是重要的事件。甚至更早,创伤后细胞代谢功能障碍是今天神经化学监测的目标。因此,必须有一种特殊的技术和人文环境,才能真正从国际比较方案监测中获益。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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