A biosocial model of the alcohol-aggression relationship.

R O Pihl, J B Peterson, M A Lau
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引用次数: 169

Abstract

Four of alcohol's dose- and rate-dependent pharmacological properties may increase the likelihood of human aggression. As an anxiolytic, alcohol is capable of reducing the inhibitory effect of fear on manifestation of aggressive behavior. As a psychomotor stimulant, alcohol can potentiate aggressive behavior, once evoked, or lower the threshold for such evocation. Alcohol-related disruption of certain higher order cognitive functions may reduce the inhibitory control generally exercised by previously established knowledge and decrease ability to plan in the face of threat or punishment. Finally, alcohol's ability to increase pain sensitivity may increase the likelihood of defensive aggression. Discussion of the nature and relevance of these pharmacological properties is structured according to a heuristic and synthetic schema, predicated upon consideration of an inhibitory neuropsychological structure--the individually and culturally determined general expectancy set.

酒精-攻击关系的生物社会模型。
酒精的剂量和速率依赖的药理学特性可能增加人类攻击的可能性。作为一种抗焦虑药,酒精能够降低恐惧对攻击行为表现的抑制作用。作为一种精神运动兴奋剂,酒精可以增强攻击行为,一旦被唤起,或降低这种唤起的阈值。酒精对某些高级认知功能的破坏可能会降低先前建立的知识通常行使的抑制性控制,并降低面对威胁或惩罚时的计划能力。最后,酒精增加疼痛敏感性的能力可能会增加防御性攻击的可能性。对这些药理学特性的性质和相关性的讨论是根据启发式和综合图式进行的,基于对抑制性神经心理学结构的考虑——个体和文化决定的一般期望集。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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