[Control of lipid metabolism in experimental obesity].

N Bégin-Heick
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引用次数: 0

Abstract

Experimental obesity syndromes are characterized by considerable fat accretion which may be the result of hyperinsulinemia. Adipose tissue accretion may be the result both of the excessive synthesis of triglycerides and a defect in their mobilization from adipose tissue stores. The adenylate cyclase system which appears to be the site of the defect in mobilization is studies in more detail.

[实验性肥胖的脂质代谢控制]
实验性肥胖综合征的特点是大量脂肪增加,这可能是高胰岛素血症的结果。脂肪组织增生可能是甘油三酯合成过度和它们从脂肪组织储存中动员缺陷的结果。腺苷酸环化酶系统似乎是在动员缺陷的地方进行了更详细的研究。
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