{"title":"Papillary meningioma.","authors":"S Z Stefanko, W M Mackay","doi":"10.1007/978-3-642-81553-9_38","DOIUrl":null,"url":null,"abstract":"<p><p>Six cases of papillary meningioma are described. Five patients are still alive 2 months, and 2, 7, 8 and 14 years after the operation respectively. No metastases or recurrences have been noted. One patient died from an intercurrent disease 2 years after operation. In all cases the papillary pattern was associated with one of the typical well known forms of meningioma. One case showed histologic features of anaplasia, i.e.: local high cellularity accompanied by mitotic activity and pleiomorphism. Only one or two features suggestive of a higher growth dynamism were noted in the other cases. The mechanism of the papillary structures is discussed. Vasotropism and a feeble cohesion between the cellular perivascular \"crowns\" are thought to be responsible for the papillary pattern which should be interpreted as a secondary phenomenon. It appears likely that the papillary pattern of meningioma is not always an expression of anaplasia when no other clinical and histologic signs of malignancy are present.</p>","PeriodicalId":75397,"journal":{"name":"Acta neuropathologica. Supplementum","volume":"7 ","pages":"126-8"},"PeriodicalIF":0.0000,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"16","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"Acta neuropathologica. Supplementum","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.1007/978-3-642-81553-9_38","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
引用次数: 16
Abstract
Six cases of papillary meningioma are described. Five patients are still alive 2 months, and 2, 7, 8 and 14 years after the operation respectively. No metastases or recurrences have been noted. One patient died from an intercurrent disease 2 years after operation. In all cases the papillary pattern was associated with one of the typical well known forms of meningioma. One case showed histologic features of anaplasia, i.e.: local high cellularity accompanied by mitotic activity and pleiomorphism. Only one or two features suggestive of a higher growth dynamism were noted in the other cases. The mechanism of the papillary structures is discussed. Vasotropism and a feeble cohesion between the cellular perivascular "crowns" are thought to be responsible for the papillary pattern which should be interpreted as a secondary phenomenon. It appears likely that the papillary pattern of meningioma is not always an expression of anaplasia when no other clinical and histologic signs of malignancy are present.