[Neck-induced vertigo].

H Scherer
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引用次数: 0

Abstract

Cervicogenic vertigo can be elicited by hyperactivity of spinovestibular afferents and, much more seldomly, by episodic reduction of blood flow in the vertebral artery. The afferent hyperactivity to the vestibular system derives from a "circulus vitiosus" involving false posture, pain, joint dysfunction, which in turn exacerbates the false posture - and so on. This is to be observed in the joints CO/C1 and C1/C2 and their short muscles. This dysfunction of the upper cervical spine can be determined by exact anamnesis, careful investigation of neck mobility, joint play and muscle tension. The cervical nystagmus observed during the neck torsion test is short (seconds) in functional diseases (hyperactivity) and longlasting (minutes) in cases of vascular disorder. Cervical vertigo should be differentiated from other atactic disorders, especially those arising from benign paroxysmal positional vertigo and from Menière's disease.

(Neck-induced眩晕)。
颈源性眩晕可由脊髓前庭传入神经的过度活跃引起,更罕见的是由椎动脉血流量的偶发性减少引起。前庭系统传入神经的过度活跃源于一种“恶性循环”,包括错误的姿势、疼痛、关节功能障碍,这反过来又加剧了错误的姿势——等等。这可以在CO/C1和C1/C2关节及其短肌中观察到。这种上颈椎功能障碍可以通过精确的记忆、仔细检查颈部活动、关节活动和肌肉紧张来确定。在颈部扭转试验中观察到的颈部眼球震颤在功能性疾病(多动)中很短(秒),在血管疾病中持续时间很长(分钟)。颈性眩晕应与其他失稳性疾病相鉴别,尤其是由良性阵发性体位性眩晕和meni病引起的眩晕。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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