Piezo1-centered mechanotransduction in knee osteoarthritis: multicompartment mechano-inflammatory networks and therapeutic opportunities

IF 4.6 2区 医学 Q1 Medicine
Jing Li,Bo Dong,Xueqin Zeng,Fei Gao,Baohui Wang
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引用次数: 0

Abstract

Abstract Knee osteoarthritis (KOA) is increasingly recognized as a whole-joint mechano-inflammatory disorder in which abnormal loading, tissue remodeling, and inflammatory sensitization reinforce one another. Piezo1 is a context-dependent mechanotransduction hub that links mechanical cues to Ca 2+ -dependent inflammatory, catabolic, cell-death, and organelle-stress responses. This focused review, supported by a structured search of PubMed, Web of Science, and China National Knowledge Infrastructure from database inception through June 2, 2026, synthesizes evidence across articular cartilage, synovium, subchondral bone, osteophyte formation, and the pain-related microenvironment. Dysregulated Piezo1-associated signaling has been implicated in extracellular matrix degradation, chondrocyte dysfunction, cell-fate imbalance, and organelle stress. Importantly, Piezo1 is not uniformly pathogenic: excessive activation can aggravate injury in mature chondrocytes and other joint cells, whereas Piezo1 activity may support reparative responses in selected progenitor-cell populations. Preclinical studies have investigated whether traditional Chinese medicine-related interventions alter components of the Piezo1-associated mechano-inflammatory network, although direct target engagement and causal validation remain limited. Artemisinin, wogonin, Shengyu Decoction, and Gubi Zhitong Formula have been associated with changes in distinct molecular modules, whereas evidence linking acupuncture and acupotomy to Piezo1 remains limited to heterogeneous preclinical models and does not establish direct or Piezo1-specific therapeutic mechanisms. However, translation remains limited by heterogeneous models, insufficient compartment- and cell-specific validation, incomplete exposure and target-engagement data, and inconsistent procedural standards. Future studies should integrate local and reversible intervention strategies, rigorous quality control, standardized procedural and stimulation parameters, and cell-specific causal validation. Piezo1 may therefore serve as a stratifiable and testable organizing axis for developing more precise KOA interventions.
膝关节骨关节炎的以压电为中心的机械传导:多室机械炎症网络和治疗机会
膝关节骨性关节炎(KOA)越来越被认为是一种全关节机械炎性疾病,其中异常负荷、组织重塑和炎症致敏相互加强。Piezo1是一个环境依赖的机械转导中枢,将机械信号与ca2 +依赖性炎症、分解代谢、细胞死亡和细胞器应激反应联系起来。这篇重点综述,由PubMed、Web of Science和中国国家知识基础设施从数据库建立到2026年6月2日的结构化搜索支持,综合了关节软骨、滑膜、软骨下骨、骨赘形成和疼痛相关微环境的证据。失调的piezo1相关信号传导涉及细胞外基质降解、软骨细胞功能障碍、细胞命运失衡和细胞器应激。重要的是,Piezo1的致病性并不一致:过度激活会加重成熟软骨细胞和其他关节细胞的损伤,而Piezo1的活性可能支持某些祖细胞群的修复反应。临床前研究已经调查了与中药相关的干预措施是否会改变与piezo1相关的机械炎症网络的组成部分,尽管直接的靶点参与和因果验证仍然有限。青蒿素、吴贡素、生瘀汤和骨痹止痛方与不同分子模块的变化有关,而针灸和针刀与Piezo1的关联证据仍然局限于异质性的临床前模型,并没有建立直接或Piezo1特异性的治疗机制。然而,翻译仍然受到异质性模型、不充分的隔间和细胞特异性验证、不完整的暴露和目标参与数据以及不一致的程序标准的限制。未来的研究应该整合局部和可逆的干预策略,严格的质量控制,标准化的程序和刺激参数,以及细胞特异性的因果验证。因此,Piezo1可以作为可分层和可测试的组织轴,用于开发更精确的KOA干预措施。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
CiteScore
8.60
自引率
2.00%
发文量
261
审稿时长
14 weeks
期刊介绍: Established in 1999, Arthritis Research and Therapy is an international, open access, peer-reviewed journal, publishing original articles in the area of musculoskeletal research and therapy as well as, reviews, commentaries and reports. A major focus of the journal is on the immunologic processes leading to inflammation, damage and repair as they relate to autoimmune rheumatic and musculoskeletal conditions, and which inform the translation of this knowledge into advances in clinical care. Original basic, translational and clinical research is considered for publication along with results of early and late phase therapeutic trials, especially as they pertain to the underpinning science that informs clinical observations in interventional studies.
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