Tissue damage at computed tomography following resolution of intracerebral hematomas.

E B Skriver, T S Olsen
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引用次数: 24

Abstract

Tissue damage as sequelae after spontaneous intracerebral hemorrhage in eight consecutive patients was investigated with computed tomography performed 3 days, 2 weeks and 6 months after the stroke. The presence of contrast enhancement after 2 weeks, hypodense areas after 6 months, and atrophy of adjacent structures were considered to be markers of irreversible tissue damage. Except for a narrow zone in the periphery of the hematoma (ring blush) contrast enhancement was not observed, neither in the hematoma nor in areas where resolution had taken place. After 6 months the site of the hematomas was not identifiable in 3 patients, and 4 patients had small 'lacunar' hypodense lesions. Only one patient had a more extensive hypodense area 6 months after the hemorrhage. The most prominent sequelae after intracerebral hematoma were atrophy of adjacent structures (i.e. atrophy of basal ganglia, thalamus and ventricular enlargement) which were seen in all patients even though irreversible de loco damage in the hematoma area was sparse or absent.

脑内血肿消退后计算机断层扫描的组织损伤。
对连续8例自发性脑出血患者进行脑卒中后3天、2周和6个月的计算机断层扫描。2周后出现对比增强,6个月后出现低密度区域,邻近结构萎缩被认为是不可逆组织损伤的标志。除了血肿周围的狭窄区域(环形红晕)外,在血肿和已经消退的区域均未观察到对比度增强。6个月后,3例患者血肿部位无法识别,4例患者出现小的腔隙性低密度病变。只有1例患者出血后6个月出现更广泛的低密度区。脑内血肿后最突出的后遗症是邻近结构的萎缩(即基底节区、丘脑和脑室增大),尽管血肿区不可逆的原位损伤很少或不存在,但所有患者均可见。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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