Shared genetic architecture between schizophrenia and subcortical brain susceptibility phenotypes.

IF 4.1 Q2 PSYCHIATRY
Yingying Xie, Jiaojiao Du, Yao Zhao, Jie Sun, Shiqi Lin, Ningnannan Zhang, Zhang Zhang, Feng Liu, Dairong Cao
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Abstract

Schizophrenia (SCZ) is a highly heritable and complex neuropsychiatric disorder. Emerging evidence implicates dysregulated brain iron, particularly in subcortical regions, in SCZ pathophysiology. Here, we systematically dissected the shared genetic architecture between SCZ and subcortical brain susceptibility phenotypes by integrating large-scale genome-wide association study (GWAS) data with quantitative susceptibility mapping (QSM) phenotypes across 16 subcortical regions. Using the MiXeR framework, we quantified the extent and pattern of shared genetic overlap between SCZ and each QSM phenotype, revealing the strongest overlap in the left and right nucleus accumbens. Conditional and conjunction false discovery rate analyses identified 666 unique shared SNPs. Functional annotation highlighted enrichment in pathways related to synaptic function and neuronal development, with pronounced expression in neurons. Additionally, we characterized their spatio-temporal expression patterns of shared genes and identified 89 significant expression-trait associations linked to SCZ-related phenotypes. Furthermore, the virtual drug screen identified 691 potential protein-drug pairs that may contribute to both iron regulation and SCZ. Our findings provide new insights into the complex interaction between subcortical brain susceptibility phenotypes and SCZ, highlighting pathways that may offer novel therapeutic strategies for SCZ and related brain susceptibility-associated conditions.

精神分裂症与皮层下脑易感性表型之间的共享遗传结构。
精神分裂症(SCZ)是一种高度遗传性的复杂神经精神疾病。新出现的证据表明,在SCZ的病理生理中,大脑铁的失调,特别是在皮层下区域。在这里,我们通过整合大规模全基因组关联研究(GWAS)数据和16个皮质下区域的定量易感图谱(QSM)表型,系统地剖析了SCZ和皮质下脑易感表型之间的共享遗传结构。使用MiXeR框架,我们量化了SCZ和每个QSM表型之间共享遗传重叠的程度和模式,揭示了左伏隔核和右伏隔核的最强重叠。条件和联合错误发现率分析确定了666个独特的共享snp。功能注释强调了突触功能和神经元发育相关通路的富集,在神经元中有明显的表达。此外,我们还分析了他们共享基因的时空表达模式,并鉴定了89个与scz相关表型相关的显著表达性状关联。此外,虚拟药物筛选确定了691对可能参与铁调节和SCZ的潜在蛋白质-药物对。我们的发现为皮层下脑易感性表型与SCZ之间复杂的相互作用提供了新的见解,突出了可能为SCZ和相关的脑易感性相关疾病提供新的治疗策略的途径。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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