Decoding glioblastoma evolution and heterogeneity through mechanistic modeling: implications for clinical translation.

IF 14.3 1区 医学 Q1 ONCOLOGY
Haowu Jiang, Wan Zhao, Hui Zhou, Rui Sun
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引用次数: 0

Abstract

Glioblastoma (GBM) is one of the most aggressive and lethal primary brain tumors in adults, characterized by dynamic clonal evolution and extensive genomic, cellular, spatial, and microenvironmental heterogeneity. Multi-omics studies have revealed that GBM follows complex evolutionary trajectories involving genetic, epigenetic, transcriptional, and immune-microenvironmental remodeling as tumors grow, adapt to the brain microenvironment, and acquire therapeutic resistance. Increasing evidence suggests that GBM may originate from aberrant neural stem or progenitor cells, including those residing in the subventricular zone, and that glioblastoma stem cells (GSCs) contribute to tumor propagation, heterogeneity, and recurrence. A key conceptual challenge is to reconcile hierarchical cancer stem cell models, in which GSCs are viewed as relatively stable tumor-propagating subpopulations, with dynamic state plasticity models, in which stem-like properties can be reversibly acquired or lost during transitions among proneural-like, mesenchymal-like, invasive, and therapy-tolerant states. Recent advances in single-cell profiling, spatial transcriptomics, lineage tracing, organoid culture, 3D bioprinting, genetically engineered models, and artificial intelligence (AI)-assisted computational modeling have substantially improved the ability to study these processes. However, no currently available model fully recapitulates human GBM heterogeneity, recurrence, treatment history, and tumor-microenvironment interactions. Therefore, model selection should be guided by clearly defined mechanistic questions rather than by reliance on any single platform. This review summarizes current advances in in vitro, ex vivo, in vivo, and computational models for studying GBM evolution and heterogeneity, and discusses how integrated model pipelines may improve preclinical drug testing, treatment-response prediction, and precision neuro-oncology.

通过机制建模解码胶质母细胞瘤的进化和异质性:对临床翻译的影响。
胶质母细胞瘤(GBM)是成人最具侵袭性和致死性的原发性脑肿瘤之一,其特点是动态克隆进化和广泛的基因组、细胞、空间和微环境异质性。多组学研究表明,随着肿瘤生长、适应脑微环境并获得治疗耐药性,GBM遵循复杂的进化轨迹,涉及遗传、表观遗传、转录和免疫微环境重塑。越来越多的证据表明,GBM可能起源于异常的神经干细胞或祖细胞,包括那些位于脑室下区的细胞,胶质母细胞瘤干细胞(GSCs)有助于肿瘤的增殖、异质性和复发。一个关键的概念挑战是协调分层癌症干细胞模型,其中GSCs被视为相对稳定的肿瘤传播亚群,与动态状态可塑性模型,其中干细胞样特性可以在前膜样,间充质样,侵袭性和治疗耐受状态之间的转换过程中可逆地获得或丢失。单细胞分析、空间转录组学、谱系追踪、类器官培养、3D生物打印、基因工程模型和人工智能(AI)辅助计算建模的最新进展大大提高了研究这些过程的能力。然而,目前还没有可用的模型完全概括人类GBM的异质性、复发、治疗史和肿瘤与微环境的相互作用。因此,模型选择应该以明确定义的机械问题为指导,而不是依赖于任何单一的平台。本文综述了研究GBM进化和异质性的体外、离体、体内和计算模型的最新进展,并讨论了集成模型管道如何改善临床前药物测试、治疗反应预测和精确神经肿瘤学。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
CiteScore
18.20
自引率
1.80%
发文量
333
审稿时长
1 months
期刊介绍: The Journal of Experimental & Clinical Cancer Research is an esteemed peer-reviewed publication that focuses on cancer research, encompassing everything from fundamental discoveries to practical applications. We welcome submissions that showcase groundbreaking advancements in the field of cancer research, especially those that bridge the gap between laboratory findings and clinical implementation. Our goal is to foster a deeper understanding of cancer, improve prevention and detection strategies, facilitate accurate diagnosis, and enhance treatment options. We are particularly interested in manuscripts that shed light on the mechanisms behind the development and progression of cancer, including metastasis. Additionally, we encourage submissions that explore molecular alterations or biomarkers that can help predict the efficacy of different treatments or identify drug resistance. Translational research related to targeted therapies, personalized medicine, tumor immunotherapy, and innovative approaches applicable to clinical investigations are also of great interest to us. We provide a platform for the dissemination of large-scale molecular characterizations of human tumors and encourage researchers to share their insights, discoveries, and methodologies with the wider scientific community. By publishing high-quality research articles, reviews, and commentaries, the Journal of Experimental & Clinical Cancer Research strives to contribute to the continuous improvement of cancer care and make a meaningful impact on patients' lives.
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