2119-P: Racism as a Shared Psychosocial Stressor in Parent–Youth Dyads: Findings from the BEAD-T1D Study

IF 9.3 1区医学 Q1 ENDOCRINOLOGY & METABOLISM

Diabetes Pub Date : 2026-06-11 DOI:10.2337/db26-2119-p

ANANTA ADDALA, SARAH HANES, LAUREN FIGG, DANIEL GARFIAS SILVA, GARY M. SHAW, DIANA NARANJO, JENNIFER RAYMOND, DAVID MAAHS, KOREY HOOD

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引用次数: 0

Abstract

Introduction and Objective: Social determinants of health, including racism and psychosocial stress, are increasingly recognized as drivers of diabetes-related outcomes. The BEAD-T1D study examined relationships in parent-youth dyads across three key domains: racism exposure, diabetes technology perceptions, and psychosocial health. Methods: Parents and youth aged 12-21 years with T1D completed validated age-appropriate surveys assessing perceived racism and adverse childhood experiences, diabetes technology acceptance and burden, and psychosocial outcomes including depression, diabetes distress, and global health. Pearson correlations were used to measure correlations within and across dyads. Results: Dyads (n=80: 50% Hispanic, 55% publicly insured) demonstrated significant within-dyad correlations on explicit racism (r=0.42, p<0.001), diabetes distress (r=0.24, p=0.034), global health (r=0.35, p=0.001), and depression (r=0.38, p=0.001). The figure presents correlation arrows between parents (♦) and youth (←) across the domains of racism exposure (purple), diabetes technology perceptions (green), and psychosocial health (blue). Conclusion: These findings demonstrate the interconnected nature of the health determinants within minoritized families. Racism exposure correlates with diabetes technology perception and psychosocial health, suggesting that racism is embedded into diabetes experience for minoritized dyads. Disclosure A. Addala: None. S. Hanes: None. L. Figg: None. D. Garfias Silva: None. G.M. Shaw: None. D. Naranjo: None. J. Raymond: None. D. Maahs: Advisory Panel; Current; Abbott Diabetes, Sanofi, Medtronic, biospex, enable biosciences, kriya. K. Hood: Consultant; Current; Sanofi, MannKind Corporation, Insulet Corporation. Funding NIDDK (5K23DK131342)

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[21] [p]种族主义作为父母-青少年共同的社会心理压力源：来自BEAD-T1D研究的结果

引言和目的：健康的社会决定因素，包括种族主义和心理社会压力，越来越被认为是糖尿病相关后果的驱动因素。BEAD-T1D研究从三个关键领域考察了父母与青少年的关系：种族主义暴露、糖尿病技术认知和社会心理健康。方法：12-21岁T1D患者的父母和青少年完成了与年龄相适应的有效调查，评估了感知到的种族主义和不良童年经历、糖尿病技术接受度和负担，以及包括抑郁、糖尿病困扰和全球健康在内的社会心理结局。Pearson相关性被用来衡量两组之间和两组之间的相关性。结果：两代人（n=80: 50%西班牙裔，55%公共保险）在显性种族主义（r=0.42, p=0.001）、糖尿病困扰（r=0.24, p=0.034）、全球健康（r=0.35, p=0.001）和抑郁（r=0.38, p=0.001）上表现出显著的两代内相关性。该图显示了父母（♦）和青年（←）在种族主义暴露（紫色）、糖尿病技术认知（绿色）和心理社会健康（蓝色）等领域之间的相关箭头。结论：这些发现表明，在少数民族家庭中，健康决定因素具有相互联系的性质。种族主义暴露与糖尿病技术感知和社会心理健康相关，表明种族主义嵌入到少数二人组的糖尿病经历中。A.阿达拉：没有。汉斯：没有。费格：没有。D. Garfias Silva：没有。通用·肖：没有。D. Naranjo：没有。雷蒙德：没有。D. Maahs：咨询小组；当前的;雅培糖尿病、赛诺菲、美敦力、biospex、enable生物科学、kriya。K. Hood：顾问；当前的;赛诺菲，曼恩公司，胰岛素公司。资助NIDDK （5K23DK131342）

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来源期刊

Diabetes 医学-内分泌学与代谢

CiteScore

12.50

自引率

2.60%

发文量

1968

审稿时长

1 months

期刊介绍： Diabetes is a scientific journal that publishes original research exploring the physiological and pathophysiological aspects of diabetes mellitus. We encourage submissions of manuscripts pertaining to laboratory, animal, or human research, covering a wide range of topics. Our primary focus is on investigative reports investigating various aspects such as the development and progression of diabetes, along with its associated complications. We also welcome studies delving into normal and pathological pancreatic islet function and intermediary metabolism, as well as exploring the mechanisms of drug and hormone action from a pharmacological perspective. Additionally, we encourage submissions that delve into the biochemical and molecular aspects of both normal and abnormal biological processes. However, it is important to note that we do not publish studies relating to diabetes education or the application of accepted therapeutic and diagnostic approaches to patients with diabetes mellitus. Our aim is to provide a platform for research that contributes to advancing our understanding of the underlying mechanisms and processes of diabetes.