Arenobufagin suppresses the progression of early-stage hepatocellular carcinoma by inhibiting EpCAM-mediated tumor stemness

Lijuan Deng, Lifang Zou, Chunhong Zhou, Fan Yang, Fen Ouyang, Zhu Y, Huihui Cao, Min Hu, Xiaoshen Zhang, Junshan Liu
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Abstract

Epithelial cell adhesion molecule (EpCAM) is a biomarker for epithelial cell-derived tumors. However, the specific role of EpCAM itself in early-stage hepatocellular carcinoma progression remains unclear, and small molecules targeting EpCAM have not yet been reported. Here, the protein expression profile of EpCAM in tumor-adjacent regions was found to be higher than that in tumor regions, and to be positively associated with the progression of early-stage liver cancer, as well as high frequency of recurrence, cirrhosis, lymph node metastasis, microvascular invasion and cancer stemness, in 68 patients with hepatocellular carcinoma (HCC). In vitro , EpCAM enhanced cancer cell stemness, as reflected by increased abilities of proliferation, self-renewal, migration and invasion, which was counteracted by arenobufagin. Furthermore, arenobufagin inhibited the viability of Hep3B and Huh7 cells with IC 50 values of 36.4 nM and 123.4 nM after 72 h of treatment, respectively. Molecular docking data further indicated that arenobufagin binds EpCAM. Moreover, arenobufagin inhibited early progression of HCC through EpCAM in a zebrafish xenograft tumor model mimicking early-stage hepatocellular carcinoma without blood vessels in vivo . This study supports a tumor-promoting role of EpCAM in early-stage hepatocellular carcinoma by facilitating cancer stemness and suggests that arenobufagin might be promising candidate for EpCAM inhibition.
arennobufagin通过抑制epcam介导的肿瘤干性抑制早期肝细胞癌的进展
上皮细胞粘附分子(Epithelial cell adhesion molecule, EpCAM)是上皮细胞源性肿瘤的生物标志物。然而,EpCAM本身在早期肝细胞癌进展中的具体作用尚不清楚,靶向EpCAM的小分子也尚未报道。本研究在68例肝细胞癌(HCC)患者中发现EpCAM蛋白在肿瘤邻近区域的表达谱高于肿瘤区域,并与早期肝癌的进展、高复发频率、肝硬化、淋巴结转移、微血管侵袭和癌干性呈正相关。在体外,EpCAM增强了癌细胞的干细胞性,表现为增强了癌细胞的增殖、自我更新、迁移和侵袭能力,而这被arennobufagin抵消了。此外,槟榔素对Hep3B和Huh7细胞的活性有抑制作用,作用72 h后ic50值分别为36.4 nM和123.4 nM。分子对接数据进一步表明,arennobufagin与EpCAM结合。此外,在模拟体内无血管的早期肝细胞癌的斑马鱼异种移植肿瘤模型中,肾蟾素通过EpCAM抑制了HCC的早期进展。本研究支持EpCAM在早期肝细胞癌中促进肿瘤的作用,通过促进肿瘤的发生,并提示arennobufagin可能是抑制EpCAM的有希望的候选药物。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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