Hemorrhagic pericardial effusion 6 months following atrial septal defect device closure: Has it eroded?

IF 0.7 Q4 CARDIAC & CARDIOVASCULAR SYSTEMS
Annals of Pediatric Cardiology Pub Date : 2025-01-01 Epub Date: 2025-07-14 DOI:10.4103/apc.apc_216_24
Alaina Zameer, Chetan Patel, Lamk Kadiyani, Damandeep Singh, Shivam Goel, Sivasubramanian Ramakrishnan
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引用次数: 0

Abstract

Transcatheter closure of atrial septal defect (ASD) is the preferred modality of treatment. Pericardial effusion occurring late after ASD device closure is extremely rare, and device-related erosion is the most likely possibility. We describe a 41-year-old male who underwent ASD device closure with a 32-mm Amplatzer Septal Occluder device and who developed cardiac tamponade 6 months after the procedure. Extensive investigations did not reveal any evidence of erosion. Given the endemicity of tuberculosis in India, exudative pericardial effusion, a positive tuberculin skin test, and the presence of a few matted mediastinal lymph nodes, the patient was started on antitubercular therapy. There was no reaccumulation of pericardial fluid at subsequent follow-ups. To conclude, not all hemorrhagic pericardial effusions following device closure of ASD are secondary to erosions. After ruling out device erosion, other etiologies for pericardial effusion should be carefully searched for.

房间隔缺损装置关闭后6个月出血性心包积液:是否有侵蚀?
经导管治疗房间隔缺损(ASD)是首选的治疗方式。ASD装置关闭后晚期出现心包积液是极为罕见的,与装置相关的糜烂是最有可能的。我们描述了一位41岁的男性患者,他接受了32毫米Amplatzer室间隔闭塞器关闭ASD装置,并在手术后6个月发生了心脏填塞。广泛的调查没有发现任何侵蚀的证据。鉴于肺结核在印度的流行,心包渗出性积液,结核菌素皮肤试验阳性,并存在一些杂乱的纵隔淋巴结,患者开始接受抗结核治疗。在随后的随访中没有发现心包积液。综上所述,并非所有ASD装置关闭后的出血性心包积液都继发于侵蚀。排除器械糜烂后,应仔细寻找心包积液的其他病因。
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来源期刊
Annals of Pediatric Cardiology
Annals of Pediatric Cardiology CARDIAC & CARDIOVASCULAR SYSTEMS-
CiteScore
1.40
自引率
14.30%
发文量
51
审稿时长
23 weeks
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