Correction to ‘CC chemokine receptor-like 1 functions as a tumour suppressor by impairing CCR7-related chemotaxis in hepatocellular carcinoma’

IF 5.2 2区医学 Q1 ONCOLOGY

The Journal of Pathology Pub Date : 2025-07-01 DOI:10.1002/path.6445

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引用次数: 0

Abstract

Jie-Yi Shi, Liu-Xiao Yang, Zhi-Chao Wang, Ling-Yan Wang, Jian Zhou, Xiao-Ying Wang, Guo-Ming Shi, Zhen-Bin Ding, Ai-Wu Ke, Zhi Dai, Shuang-Jian Qiu, Qi-Qun Tang, Qiang Gao* and Jia Fan*. J Pathol 2015; 235: 546–558. https://doi.org/10.1002/path.4450

The authors apologise for any inconvenience this error may have caused.

While considering the request to make a correction, the editors realised that two of the seven cell lines used in the article are listed as problematic in Cellosaurus (https://www.cellosaurus.org/). When the studies were performed, the cell line SMMC-7721 was considered to be an hepatocellular carcinoma cell line, and HL-7702/L02 was considered to originate from normal foetal liver; both are listed in Cellosaurus as derivatives of HeLa (a human papillomavirus-related endocervical adenocarcinoma cell line). Limited use was made of these two cell lines in the article, but readers should consider the possibility that they were contaminated.

Abstract Image

查看原文本刊更多论文

更正“CC趋化因子受体样1通过损害肝细胞癌中ccr7相关的趋化性而发挥肿瘤抑制作用”。

石杰毅，杨柳晓，王志超，王凌燕，周健，王晓英，石国明，丁振斌，柯爱武，戴智，邱双建，唐启群，高强*，樊佳*。中华病理学杂志2015；235: 546 - 558。https://doi.org/10.1002/path.4450作者对这个错误可能造成的任何不便表示歉意。在考虑进行更正的请求时，编辑们意识到，文章中使用的七个细胞系中有两个在Cellosaurus （https://www.cellosaurus.org/）中被列为有问题的细胞系。在进行研究时，细胞系SMMC-7721被认为是肝细胞癌细胞系，HL-7702/L02被认为起源于正常胎儿肝脏；两者都在celllosaurus中被列为HeLa（一种人乳头瘤病毒相关的宫颈内膜腺癌细胞系）的衍生物。文章中对这两种细胞系的使用是有限的，但读者应该考虑到它们被污染的可能性。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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来源期刊

The Journal of Pathology 医学-病理学

CiteScore

14.10

自引率

1.40%

发文量

144

审稿时长

3-8 weeks

期刊介绍： The Journal of Pathology aims to serve as a translational bridge between basic biomedical science and clinical medicine with particular emphasis on, but not restricted to, tissue based studies. The main interests of the Journal lie in publishing studies that further our understanding the pathophysiological and pathogenetic mechanisms of human disease. The Journal of Pathology welcomes investigative studies on human tissues, in vitro and in vivo experimental studies, and investigations based on animal models with a clear relevance to human disease, including transgenic systems. As well as original research papers, the Journal seeks to provide rapid publication in a variety of other formats, including editorials, review articles, commentaries and perspectives and other features, both contributed and solicited.